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Sex & Dyspareunia

Sex and Dyspareunia

The term dyspareunia, difficult or painful coitus, has always been presumed to refer to coital distress in women. The word stems from the Greek, and somewhat freely translates into “badly mated.”

Since no comparable word reflecting or suggesting coital distress for men has been established, poetic license will be begged. Here is comprised of two separate sections devoted to consideration of individual complaints of female and male sexual dysfunction identified by the individuals involved as difficult or painful coitus. Men can be “badly mated” too!

That factor in the total of male and female sexual dysfunction perhaps most difficult for the therapist to define involves the psycho physiological complaint of dyspareunia. Diagnostic insecurity relates directly to the fact that dyspareunia has a varied number of both subjective and objective origins that frequently give rise to combinations of psycho physiological distress rather than complaints that can be categorized individually.

Avoid Sex

For years, woman’s complaint sex hurts when had intercourse has been an anathema to the therapist. Even after an adequate pelvic examination, the therapist frequently cannot be sure whether the patient is complaining of definitive but undiagnosed pelvic pathology or whether, as has been true countless thousands of times.

A sexually dysfunctional woman is using the symptomatology of pain as a means of escaping completely or at least reducing markedly the number of unwelcome sexual encounters in her marriage.

For it is true that once convinced that there is no recourse for reversal of his or her dysfunctional status, the sexually inadequate partner in any marriage manufactures excuse after excuse to avoid sexual confrontation.

As women have long since learned, a persistent, aggressive male partner can overwhelm, neutralize, or even negate the most original of excuses to avoid sexual exposure.

However, presuming any degree of residual concern for or interest in his partner as an individual, the husband is rendered powerless to support his insistence upon continuity of sexual contact when the wife complains of severe distress during or after sexual connection.

If the female partner complains and flinches with penile insertion, moans and contracts her abdominal and pelvic musculature during the continuum of male thrusting, cries out or screams with deep vaginal penetration, sheds bitter tears after termination of every sexual connection, or complains angrily of aching in the pelvis or burning in the vagina during or even hours after a specific coital episode.

The male sexual approach must be accepted as the probable potentiator of a physiological basis for his female partner’s evidenced sexual dysfunction. Thereafter, the husband has minimal recourse. There is little he can do other than to avoid or at least reduce marital-unit sexual exposure on his own cognizance, and/or to insist that his wife seek professional consultation.

Once consulted, the twofold problem that constantly baffles authority is first whether a specific physiological basis can be defined for the objective existence of pain. Second, if not, whether the existence of pelvic pathology should arbitrarily be ruled out, thereby defining the registered complaint of dyspareunia as subjective in origin. When a woman complains of pain during or after intercourse, there are very few diagnostic landmarks to follow for treatment, so that consideration of the etiology of the painful response seems appropriate.

As in vaginismus, a differential diagnosis cannot be established for a complaint of dyspareunia unless careful pelvic and rectal examinations are conducted. Even then there can be no sure diagnosis if the existence of pelvic pathology is denied purely on the basis of negative examinations by competent authority.

Yet, in a positive vein, there are obvious pelvic or rectal findings that can and do support objectively a woman’s subjective complaint of coital discomfort. The female partner’s persistent complaint of pain with any form of coital connection must not be authoritatively denied or, for that matter supported, purely on the basis of interrogation, regardless of how carefully or in what depth the questioning has been conducted.

There are many varieties of dyspareunia, varying from postcoital vaginal irritation to severe immobilizing pain with penile thrusting. Symptomatic definition relating not only to the anatomy of the vaginal barrel but also to the total of the reproductive viscera is in order.

In no sense will the discussion include all possible forms of pelvic distress. Considered, however, will be the major sources of pelvic pathology engendering painful response from the female partner during or after coital connection. The dyspareunia will be considered in relation to specific areas of the vaginal barrel, the reproductive viscera, and the soft tissue components of the pelvis, and to painful stimuli developing, in a time-related sequence during or after coital connection.

Sex and Painful Vagina

The complaint of pain with penile intromission should demand clinical inspection of the vaginal outlet and the labial (major and minor) area. Direct observation can easily delineate any of the following minor areas of concern, minor only in the sense of easy reversibility of physical distress by adequate clinical measures.

An intact hymen or the irritated or bruised remnants of the hymenal ring can and do cause outlet pain during attempted coital connection. Less obvious is an unprotected scar area just at the mucocutaneous juncture of the vaginal mucosa and the perineal body.

These scars, primarily residuals of episiotomies sustained during childbirth, occasionally have been observed to result from criminal abortion techniques or gang-rape episodes. The Bartholin-gland area in the minor labia should be carefully palpated for enlargement in the gland base, which can contribute to a locally painful reaction as the vaginal outlet is dilated by the penile glans at onset of intromission.

Finally, in postmenopausal women the labia and vaginal outlet may have so lost elasticity and become so shrunken in size that any penile insertive attempt will return a painful response.

Sex and Clitoris Irritation

With any complaint of outlet pain, the clitoral area also should be inspected carefully. Many women simply cannot define anatomically or are too embarrassed to discuss objectively the exact location of the outlet distress occasioned by attempts at coital connection.

Smegma beneath the clitoral foreskin can cause chronic irritation and burning that becomes severe as the penis is introduced into the vaginal orifice. Rarely adhesions beneath the minor labial foreskin anchoring the foreskin to-the clitoral glans can cause distress when the foreskin is moved or pulled from its specific pudendal-overhang position by manipulative approaches to the mons area or by intromissive attempts.

When the minor labial hood of the clitoris is pulled down toward the perineum by the act of penile intromission, an intense pain response from the presensitized clitoral glans or even the clitoral shaft may become of major clinical moment.

The same type of reaction can be elicited if foreplay in the clitoral area has been irritative rather than stimulative in character, as so often happens when the sexually uneducated male tries to follow “authoritative” directions in attempts to stimulate his partner sexually. Heavy handed manipulation or frequent masturbatory irritation can elicit painful responses from the clitoral-glans area. This irritative reaction may develop rapidly

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Sex & Dyspareunia

Pelvic Disease

Endometriosis is a disease in which implants of endometrial tissue spread throughout the pelvic viscera and their protective covering, the peritoneum. When examined microscopically, this ectopic tissue resembles the lining of the inner cavity of the uterus.

The tubes, ovaries, broad ligaments, omentum, and the posterior wall of the uterus may be involved by firm fibrous adhesions. There are even many instances of tying together omentum and bowel with the reproductive viscera into large pelvic masses. The etiology of endometriosis has not been fully established.

It would not serve the purposes of this text to enter into a detailed discussion of the subject. Although endometrial implants appear in many anatomical areas other than the pelvic viscera, consideration will be focused alone on local pelvic implants.

Even if there are no major adhesions in the pelvis, there are at least minor elements of continuous local peritoneal irritation. Endometrial nodules usually can be felt most effectively with simultaneous manual pelvic-rectal examination.

The pain created by intercourse is due to the constriction and immobilization of the peritoneum and the firming up of the soft tissues of the pelvis by adhesions. The pelvic structures have progressively less facility to distend, expand, and move freely as the endometriosis progresses.

There is consequently more local tissue resistance to involuntary vaginal expansion, uterine elevation, and male pelvic thrusting.

In all situations that create chronic irritation of the pelvic peritoneum, fixation of the uterus, or constriction of the vaginal barrel, pain with intercourse is a relatively constant finding.

Treatment for endometriosis is either medical or surgical depending upon the degree of soft-tissue and pelvic visceral involvement. But once endometriosis has developed to a point at which there is significantly severe pain in response to coital activity, there must be definitive treatment of the condition, or the individual woman will have little hope of relief from the symptoms of progressively increasing dyspareunia.

Post Surgical

There are three important sources for acquired dyspareunia following removal of the uterus for specific organ pathology. First, dyspareunia results from thoughtless surgical technique. Physicians, when performing a hysterectomy, may overlook the fact that the cervix enters the vagina through the superior wall of that organ. When the wound in the vaginal barrel is repaired after removal of the cervix, if care is not taken to retain a superior position for the vaginal cuff, the scarred area, instead of being retained in the superior vaginal wall, may be pulled into the depth of the barrel by tissue constriction or by excessive folding or removal of vaginal tissue.

Postoperatively when the husband thrusts deeply into the vagina, the penis can come into contact with the resistant scarred area. There is little residual facility for involuntary vaginal distention in the area of the surgical scar.

Therefore, dyspareunia of significant proportion develops occasionally as a post surgical complication. Since this unfortunate result usually does not develop for months or even a year after surgery, the operating surgeon may never be made aware of the acquired dyspareunia.

The second opportunity to acquire dyspareunia is occasioned by the surgical indications for removal of the ovaries at the time the uterus is removed, or for that matter, at any time. If post operative sex-steroid-replacement is not initiated, many women will develop senile changes in the vagina and, in time, secondary dyspareunia.

The third incidence of dyspareunia after hysterectomy rarely comes to the attention of the operating surgeon. The etiology of the acquired dyspareunia may be subjective in origin.

Sexual Anxious Woman

If the woman facing hysterectomy and/or removal of the ovaries is not reassured with her husband that there need not be reduction of sexual drive or orgasmic facility after surgery, her fantasy and her friends’ old wives’ tales may, by power of suggestion, create fears of sexual performance for the anxious woman.

If she feels that she is going to be castrated, and sex-steroid-replacement therapy is not explained and offered as indicated, she well may believe that after surgery there will be loss of ability to respond in a sexually effective manner in the future. What is worse, an uninformed husband may have similar concepts.

If anything, sexual responsivity should be higher shortly after than immediately before surgery. The pelvic pathology for which the hysterectomy or oophorectomy is indicated usually detracts from sexual effectiveness by creating a state of ill health which, in turn, reduces innate sexual tension.

When the offending condition is removed and the general state of health consequently improved, there usually is a reawakening of sexual interest. If women are not reassured before surgery, many presume that, in the future, intercourse will provide no return for them or for their husbands, or that intercourse will even be painful.

Any woman has only to be sure that she will be distressed by future coital connection to take a long step toward acquired dyspareunia.

There are, of course, many factors other than the major ones of infection, endometriosis, post surgical objective and subjective complications, and the syndrome of broad-ligament laceration that create painful stimuli from irritated peritoneal and pelvic soft tissues in response to coital connection.

These include tumors of the uterus, such as myomas (fibroids), ovarian cysts and solid tumors, and, carcinoma of the female reproductive tract. Any of these tumor growths occasionally incite onset of the complaint of acquired dyspareunia. Those interested can find more definitive evaluations of this physiological source of dyspareunia in current gynecology textbooks.

Thus, the basic premise with which the Foundation approaches the problem of dyspareunia is one of elimination of possible pathological reasons for the complaint. If a woman complains of pain with intercourse, her complaint is accepted at face value, and steps are taken to identify the biophysical source of the coital distress.

The diagnosis of psychosomatic dyspareunia, unquestionably of moment in the sexual-response field, must be made by exclusion. To assign subjective origins to pelvic pain, regardless of the patient’s personality structure, without definitive physical evaluation of the pelvis, can result in clinical mismanagement of patients. Certainly there are times when, after every effort has been made to establish physical source of the pelvic pain, subjective etiology for the complaint will be considered strongly. But the initial bio physical investigative effort must be made by competent authority.

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Sex & Dyspareunia

Curve Penis

Peyronie
A disease produced by induration and fibrosis of the corpora cavernosa of the penis and evidenced as an upward bowing of the penis, plus a gradually increasing angulation to the right or left of the midline, makes coital connection somewhat difficult, and in advanced stages coition is virtually impossible.

There also may be pain attached to attempts at coital connection due to the unusual angulation of the penis creating resultant penile shaft strain, both with inserting and with thrusting experience.

Penile Chordee or Curved Penis

It is seen rarely in situations of penile trauma and only occasionally with neglected gonorrheal urethritis. Consultation has been requested by four men with severe penile chordee as a post traumatic residual.

In two instances the fully erect penis was struck sharply by an angry female partner. The remaining two men each described severe pain with a specific coital experience. During uninhibitedly responsive coital connection with the female partner in a superior position, the penis was lost to the vaginal barrel. In each case, the women tried to remount rapidly by sitting down firmly on the shaft of the penis.

The vaginal orifice was missed in the hurried insertive attempt and the full weight of the woman’s body sustained by the erect penis.

Each of the four men gave the remarkable verbal description that he felt or heard something snap. Shortly thereafter an obvious hematoma appeared on the anterior or posterior wall or lateral walls of the penile shaft.

Over a period of weeks, as the local hemorrhage was absorbed, fibrous adhesions developed and, with subsequent scar formation, there slowly developed a downward bowing and (in three cases) mild angulation of the penis.

Urologists state that due to the type of tissue involved in the penile trauma, there is little to offer in the way of clinical reprieve for men afflicted with these embarrassing erective angulations, Peyronie’s disease or chordee.

Attempts at surgical correction currently are of relatively little value and not infrequently make the situation worse. Any of these situations create responses of pain and tenderness during both masturbation and coital connection.

It always should be borne in mind that the erect penis can be traumatized by a sudden blow, by rapidly shifting coital position, by applying sudden angulation strain to the shaft, or from violent coital activity that places sudden weight or sudden pressure on the fully erect penis. The unfortunate residuals of such trauma have been described above.

Direct trauma of the penis occasioned by major accidents, war injuries, or direct physical attack sometimes requires that treatment for sexual dysfunction be patterned to include marked variation in the anatomical structuring of the penis. In anatomical deformity of the penis, the complaint of dyspareunia can be raised by either the male or female sexual partner.

Testicular Pain

Usually of the dull, aching variety, develops for some men who spend a significant amount of time in sexual play or in reading pornographic literature, concurrently maintaining erections for lengthy periods of time without ejaculating within the immediate present.

Frequent returns to excitement or even plateau-phase levels of sexual stimulation without ejaculatory relief of the accompanying testicular vasocongestion can cause an aching in either or both testes, particularly in younger men. Relief is immediate with ejaculation, which disperses the superficial and deep vasocongestion and returns the testicles to their normal size.

No permanent damage is occasioned by maintaining chronic testicular congestion for a period of days. Men with this syndrome of testicular pain occasioned by long-maintained sexual tension are in the minority.

Usually, the syndrome of involuntary testicular pain is relieved somewhat as the man ages.

There are painful reactions that develop during or shortly after coital connection that particularly reflect the influence of the vaginal environment. These situations are mentioned only in passing, but the therapist should keep in mind the fact that the basic pathology involved rests within the vaginal environment.