Categories
Overall Health

How Do I Check My Blood Sugar Level?

Follow your doctor’s advice and the instructions that come with the glucose meter. In general, you will follow the steps below. Different meters work differently, so be sure to check with your doctor for advice specifically for you.

  1. Wash your hands and dry them well before doing the test.
  2. Use an alcohol pad to clean the area that you’re going to prick. For most glucose meters, you will prick your fingertip. However, with some meters, you can also use your forearm, thigh, or the fleshy part of your hand. Ask your doctor what area you should use with your meter.
  3. Prick yourself with a sterile lancet to get a drop of blood. (If you prick your fingertip, it may be easier and less painful to prick it on one side, not on the pad.)
  4. Place the drop of blood on the test strip.
  5. Follow the instructions for inserting the test strip into your glucose meter.
  6. The meter will give you a number for your blood sugar level.

Tips on blood sugar testing

  1. Pay attention to expiration dates for test strips.
  2. Use a big enough drop of blood.
  3. Be sure your meter is set correctly.
  4. Keep your meter clean.
  5. Check the batteries of your meter.
  6. Follow the instructions for the test carefully.
  7. Write down the results and show them to your doctor.

How often should I check my blood sugar level?

Check your blood sugar as often as your doctor suggests. You’ll probably need to do it more often at first. You’ll also check it more often when you feel sick or stressed, when you’re changing your medicine or if you’re pregnant. People taking insulin may need to check their levels more often.

Keep track of your blood sugar levels by writing them down. You can also keep track of what you’ve eaten and how active you’ve been during the day. This will help you see how food and exercise affect your blood sugar level.

What should my blood sugar level be?

Talk with your doctor about what is a healthy blood sugar level range for you. A level of 80 to 120 before meals is often a good goal, but not everyone who has diabetes can get their blood sugar level this low.

Be sure to talk with your doctor about what to do if your blood sugar level isn’t within the range that’s best for you.

How does food affect my blood sugar level?

Anytime you eat, you put sugar in your blood. Eating the right way can help control your blood sugar level.

You need to learn how what you eat affects your blood sugar level, how you feel, and your overall health. As a general rule, just following a healthy diet is wise. Your doctor may suggest you meet with a dietitian who can teach you how to make healthier food choices.

Categories
Senior Health

Erectile Dysfunction In Aging Male

Composite case studies have been selected to identify and illustrate the dysfunctional characteristics of the male aging process.

Both Mr. and Mrs. A were 66 and 62 years of age when referred to the Foundation for sexual inadequacy. They had been married 39 years and had three children, the youngest of whom was 23 years of age. All children were married and living outside the home.

They had maintained reasonably effective sexual interchange during their marriage.

Mr. A had no difficulty with erection, reasonable ejaculatory control, and, aside from two occasions of prostitute exposure, had been fully committed to the marriage. Mrs. A occasionally orgasmic during intercourse and regularly orgasmic during her occasional masturbatory experiences had continued regularity of coital exposure with her husband until five years before referral for therapy.

Mr. A had recently retired from a major manufacturing concern. He had been relatively successful in his work and there were no specific financial problems facing man and wife during their declining years.

Both members of the marital unit had enjoyed good health throughout the marriage. At age 61, he had taken his wife abroad on a vacation trip which entailed many sightseeing trips with a different city on the agenda almost every day.

They were chronically tired during the exhausting trip, but because they were on vacation and away from home there was a definite increase over the established frequency of coital connections. Mr. A noted for the first time slowed erective attainment.

Regardless of his level of sexual interest or the depth of his wife’s commitment to the specific sexual experience, it took him progressively longer to attain a full erection. With each sexual exposure his concern for the delay in erective security increased until finally, just before termination of the vacation trip, he failed for the first time to achieve an erection quality sufficient for vaginal penetration.

When the coital opportunity first developed after return home, erection was attained, but again it was quite slow in development. The next two opportunities were only partially successful from an erective point of view, and thereafter he was secondarily impotent.

After several months they consulted their physician and were assured that this loss of erective power comes to all men as they age and that there was nothing to be done. Loath to accept the verdict, they tried on several occasions to force an erection with no success. Mr. A was seriously depressed for several months but recovered without apparent incident.

Approximately 18 months after the vacation trip, the couple had accepted their “fate.” The impotence was acknowledged to be a natural result of the aging process. This resigned attitude lasted approximately four years.

Although initially the marital unit and their physician had fallen into the socio-cultural trap of accepting the concept of sexual inadequacy as an aging phenomenon, the more Mr. and Mrs. A considered their dysfunction the less willing they were to accept the blanket concept that lack of erective security was purely the result of the aging process.

They reasoned that they were in good health, had no basic concerns as a marital unit, and took good care of themselves physically. Therefore, why was this dysfunction to be expected simply because some of their friends reportedly had accepted the loss of male erective prowess as a natural occurrence?

Each partner underwent a thorough medical checkup and sought several authoritative opinions, refusing to accept the concept of the irreversibility of their sexual distress. Finally, approximately five years after the onset of a full degree of secondary impotence, they were referred for treatment.

Sexual functioning was reconstituted for this marital unit within the first week after they arrived at the Foundation and as soon as they could absorb and accept the basic material directed toward the variation in the physiological functioning of the aging male.

No longer were they concerned with the delay in erective attainment; there were no more attempts to will, force, or strain to accomplish erection under assumed pressures of performance.

In short:
They needed only the security of the knowledge that the response pattern which initially had raised the basic fear of dysfunction was a perfectly natural result of the involutional process.

When they could accept the fact that it naturally took longer for an older man to achieve an erection, particularly if he were tired or distracted, the basis for their own sexual inadequacy disappeared.

Some six years after termination of the acute phase of therapy, this couple, now in the early seventies and late sixties, continue coital connection once or twice a week.

The husband has learned to ejaculate on his own demand schedule, and neither partner attempts a rapid return to sexual function after a mutually satisfactory sexual episode.

Husband and wife B

The husband, age 62, and his wife, age 63, were referred to the Foundation. They had two children, both of whom were married and lived out of the home.

Their sexual dysfunction had begun when the husband was 57 years old. He had noted some delay in attaining erection and marked reduction in ejaculatory volume and was particularly concerned with the fact that the ejaculatory experience was one of the mere dribbling of seminal fluid from the external urethral meatus, under obviously reduced pressure.

All these involutional signs and symptoms developed within approximately a year after he had noticed some delay in onset of erection attainment.

The more he worried about his symptoms the more frequent the occasions of impotence.

Mrs. B was completely convinced that this pattern of sexual involution was true to be expected as part of the aging process.

Rather than distress her husband, she suggested that they use separate bedrooms.

She changed from a pattern of free and easy exchange of sexual demand to one of availability for coital connection only at her husband’s expression of interest. To resolve her own sexual tensions, she masturbated about once every ten days to two weeks without her husband’s knowledge.

Finally, Mr. B developed severe prostatic spasm with ejaculation during approximately half the increasingly rare occasions when there was sufficient erective security to establish a coital connection. It was the persistence of this symptom of pain that first brought medical consultation and ultimately referral for treatment.

In the evaluation of this man during the physical examination, there was marked muscular weakness noted, a history of easy fatigability, and increasing lassitude in physical expression.

Mr. B also had been distressed in the last two to three years before referral to therapy with distinct memory loss for recent events. He described the loss of work effectiveness for approximately the same length of time.

With these overt symptoms suggestive of steroid starvation, testosterone replacement was initiated empirically. Within those days there was a partial return of ejaculatory pressure and a moderate shortening of the time span for the delayed erective reaction. The prostatic pain did not recur.

Husband and wife B Steroid Replacement

Once Mrs. B could accept the explanation for the onset of her husband’s sexual dysfunction, she was pleased to return to the role of an active sexual partner. The coital connection has continued regularly for the past three years with both members of the marital unit supported by steroid-replacement techniques.

In brief, for the sexually dysfunctional aging male, the primary concern is one of education so that both the man and his wife can understand the natural involutional changes that can develop within their established pattern of sexual performance.

Sex steroid replacement should be employed only if definite physical evidence of the male climacteric exists. As the newer techniques for establishing testosterone levels in blood serum become more widely disseminated, it will be infinitely easier to define and describe the male climacteric and therefore to offer testosterone replacement to those who need it, on a more definitive basis than an empirical diagnosis.

With effective dissemination of information by proper authority, the aging man can be expected to continue in a sexually effective manner into his ninth decade. Fears of performance are engendered by a lack of knowledge of the natural involutional changes in male sexual responsivity that accompany the aging process.

Really, the only factor that the aging male must understand is that loss of erective prowess is not a natural component of aging.

Statistical evaluation of the aging population and a consideration of treatment failure rates will constitute the following section. This material arbitrarily has been placed in the male rather than the female.

First, it was felt important to keep the statistical consideration of the aging marital units together, and second, because there were 56 males So years old or over in marital units accepted for treatment and only 37 wives 50 or older, it seemed appropriate to include the brief statistical discussion in the chapter reflecting the larger segment of the aging population.

Categories
Men's Health

The 11 Most Common Sexually Transmitted Diseases

The most important fact of sexually transmitted diseases is they are not contracted by people who have only one partner.

At the Clinic

Some clinics which specialize in the diagnosis and treatment of sexual diseases are free. Others charge for their services. Some are walk-in; others require an appointment to be made first. Many women opt to visit a health clinic rather than a family physician. They prefer the anonymity of their surroundings. When the infection is cured, there is no record in the family files.

Some STDs are “notifiable.” By law, they must be reported to the local health authorities. This varies from area to area, and from time to time. Other STDs are anonymous; a number instead of a name is used. Still, other diseases are confidential; name, address, and telephone number are kept in secret files. Again, this varies with the area and the time.

Some STDs have more than one name. Others have their names changed as more is discovered about them. They then get placed in their own special category; they no longer belong to the group they were originally designated. In much the same way, therapies and medications vary from clinic to clinic and from time to time. Though this can seem confusing, it shows an increase in medical knowledge of the disease. Also, environmental conditions and the endemic nature of the infection in one particular area are taken into account.

In towns and cities, there are hotlines to call for advice, help, and information. There are telephone tapes which are useful too. In isolated areas, look for notices in public locales, such as town halls, libraries, and restrooms. Consult the phone book. Entries might be under V for venereal disease or $ for STD. Above all, avoid delay in seeking help.

High Risk Behaviors

High risk sexual behavior includes:

  • Sex which is paid for.
  • Constant change of heterosexual partners.
  • Heterosexual anal sex which is unprotected.
  • Sex with an intravenous drug user.
  • “Tough” sex which causes lesions, bruises, bleeding.
  • Male sex (anal homosexual intercourse).

AIDS is transmitted by the HIV virus in blood, semen, and vagina fluids. It can be passed in skin sores and genital lesions too tiny to be seen with the unaided eye. It is also passed from mother to child in breast milk. Infected blood and semen contain the highest concentration of the virus. Vagina fluids have a lesser concentration. HIV may be present in sweat, saliva, and tears, but the concentrations are usually too weak for there to be any risk.

STDs, however, pass in very low concentrations. One germ can be enough. Studies suggest that syphilis and herpes are significant risk factors in the transmission of HIV. The sores of either disease can be on the mouth or inside the rectum, as well as on the genitals. In women, HIV is linked with a history of genital warts. It seems likely that STDs, which disrupt epithelial (lining) tissue, are important factors in the transmission of HIV. An appropriate way to avoid infection is to avoid direct contact with a partner’s semen, blood, or sores anywhere on the skin. Condoms provide some protection.

Gonorrhea

Gonorrhea is a bacterial infection which affects one million people each year in the United States. It is believed a further one million cases each year go unreported, because the disease is asymptomatic in 10 to 15 percent of men, and in 50 to 80 percent of women. Of the women with mild symptoms, 40 to 60 percent ignore them, believing that they are due to some other minor problem. The cervix is the most common site of gonorrhea.

Symptoms appear 3 days to 2 weeks after sexual contact. There is a thick, yellowish discharge. The cervix looks red, with small bump-like pits which are erosions. The urine tract often becomes infected, with the classic symptoms of UTI: stinging pain, frequency, and urgency. The infection can spread, to Skene’s and Bartholin’s glands. With oral sex, gonorrhea can spread from the penis to the throat, with sore throat and swollen glands, or it is asymptomatic. Discharge from an infected vagina or anal sex can infect the rectum with itching anus and discharge.

Untreated gonorrhea can lead to pelvic inflammatory disease (PID). Some 1 to 3 percent of women develop “disseminated gonorrhea,’ which spreads throughout the system. It can cause arthritis and, in rare cases, heart disease. The infection can be passed to a baby during birth, causing serious infection and possible blindness. Therapy is by antibiotics. Protect the cervix.

Syphilis

The corkscrew shaped bacteria of syphilis penetrate the skin of the vulva and within 30 minutes reach the glands in the groin. Thirty-six hours after infection, the bacteria have doubled in number. They double again every 30 hours. It takes an average 3 weeks (10 to 50 days) for the first symptoms to appear. By then, there are countless bacteria in the blood stream.

The first symptom is a chancre, an ulcer which starts as a pimple and then develops into an open sore with a hard rim. It is painless and self-healing. Once the sore disappears, bacteria travel in the blood, rapidly multiplying. Second stage syphilis occurs 2 to 6 weeks later. The symptoms include a skin rash over the body, swollen glands, and a flu-like condition; but often the disease is asymptomatic. Syphilis continues to wreak its havoc in the vital organs. In later years, the tertiary (third) stage is devastating: heart and brain disorders, joint inflammation, and sometimes early death.

Only about 10 percent of women who get chancres notice them. They can be hidden in the folds of the labia, under the hood of the clitoris, inside the vagina or rectum, on the cervix itself. The bacteria enter through any tiny skin lesion. The sores can appear anywhere, the most usual places being the mouth, nostril, tongue, even the finger. Avoid sexual contact if sores appear on any skin parts. The same applies to a partner.

Antibiotics destroy the bacteria of syphilis. Regular blood tests are necessary for the next two years to check for lingering germs. Keep all follow-up appointments to ensure that the disease has finally gone. Syphilis is 3 times more common in men than women; it is rare in female homosexuals. It can be passed to the fetus after the 20th week of pregnancy, so a blood test for syphilis is now a routine part of prenatal care.

Anal Sex

Anal sex carries specific health risks for all lovers, be they heterosexual or homosexual. Faeces contain highly infectious matter. The walls of the rectum are only a few cells thick. They are not designed to resist the pressure of a thrusting penis. They tear easily, and microscopic bleeding occurs. If the penis is not washed immediately after anal sex, whatever germs are in the bowel are thrust directly into the vagina. Infected semen, blood, or faeces can then pass directly into the blood system. Repeated attacks of yeast overgrowth can also occur this way.

Whatever the moral stance, hygiene is top priority. The penis should not touch the vulva, nor should it ever enter the vagina straight from the bowel. Hands, particularly fingernails, are an added danger in anal sex. Wiping with a tissue is not enough. Penis, hands, mechanical toys, all must be thoroughly scrubbed. It is strongly recommended a condom be used during anal sex, and immediately discarded afterwards.

Oral Sex

Specific micro-organisms inhabit the mouth, just as they inhabit other body orifices (openings). They rarely cause problems within their natural ecology. If they are transmitted to other orifices, they can cause infection. One typical example is a harmless bacteria of the mouth which can come in contact with the penis. The germs enter the urinary tract, and cause male UTI.

The membranes which line the mouth are naturally subjected to tiny lesions. It has been estimated that there is gum bleeding after brushing the teeth in at least one-third of any given population. Small ulcers can be present at the sides of the mouth. The tongue can be sore for a variety of reasons. All these factors can make the mouth an “unsafe” place for sex.

Diseases known to be transmitted by oro-genital infection are: the herpes virus cold sore, yeast infections, AIDS, gonorrhoea of the throat, and syphilis chancre of the lips. At least two cases of AIDS have been contracted this way. It would seem unlikely that a woman would wish to kiss a partner with a sore on the mouth, or that she would perform oral sex on a penis with a “drip”. Yet all infections have an incubation period. There is a time lapse between contracting a disease and the appearance of symptoms. Incubation periods vary widely with different STDs; they can take years for AIDS. With a new partner, the incubation period must be taken into account.

In some cases, both partners are asymptomatic. There are no signs of disease to remind lovers that oral sex can be hazardous. Avoid direct mouth contact with semen. Where there is high risk sexual activity, one option is to completely avoid oro-genital sex. If this is unacceptable, wait until a new partner has been tested and is known to be infection-free.

Tricky Trichomonas

Trichomonas vaginalis, or trich, is caused by a one-celled protozoan which grows rapidly within the vagina. Some women have an immediate and painful reaction to trich. Many more have asymptomatic trich; it is often only found if there are tests for other problems. The symptoms include a thin, foamy discharge which is yellow, green, or grey; there is intense itching and soreness, especially if the vulva is scratched. Trich can infect the urinary tract, causing burning, urgency and frequency. No tiny, one-celled creature should be able to cause such misery. But it does.

Trich can be passed on damp material: towels, bathing suits, washcloths, and toilet seats. This is rare. In most cases, it is transmitted by direct sexual contact. Metronidazole in Flagyl destroys trich. It has side effects, and should not be taken if there is any risk of pregnancy. A partner must be treated. Eschew douches and tampons. Avoid a flare-up recurrence by following the same “cool and dry” regime as for yeast overgrowth.

Vaginitis

There are many other organisms that can attack the area. They come under the generic terms nonspecific vaginitis and vulvitis. Nonspecific refers to conditions in which the cause is uncertain. They may be due to sexual infection, or they may not. The symptoms are often the same as for yeast and trich, with a profuse, foul-smelling discharge, intense itching, soreness, and in some cases, severe pain. Again, like yeast and trich, none of these attacks seem to affect the cervix. Yet they can cause real misery, and greatly reduce the quality of life.

Have a test for diabetes or a prediabetes condition first. Check diet and general health; try to boost the immune system by getting more rest, more profound sleep. Many women are run down and exhausted without realizing how deeply tired they are. Once yeast and trich are ruled out, a course of antibiotics may be the answer, though yeast overgrowth may then have to be treated. If attacks of vaginitis or vulvitis do recur, be extra scrupulous with genital hygiene. Keep the entire area cool and dry.

Chlamydia

Chlamydia is the most common STD in the U.S. today, with as many as 4 million new cases each year. It causes about half the known cases of NGU (non-gonococcal urethritis) in men. It breeds on the cervix in women. The symptoms are often mild and frequently go unnoticed. They are the same symptoms as for gonorrhea and can be confused with it. However, they appear a little later, within 1 to 3 weeks of sexual contact. More rarely, Chlamydia can be passed by a hand infected with the discharge from parent to baby.

If left untreated, chlamydia can lead to PID and infertility. Tests involve taking swabs from the cervix and culturing a specimen. The antibiotic of choice is tetracycline. Protect the cervix.

Herpes

The first attack of the herpes virus is the most painful and takes the longest time to heal. Within 2 to 20 days after infection, there is a mild tingling or itching. This can be on the labia, clitoris, or vagina opening; more rarely on the vagina wall, the cervix, the buttocks, thighs, or anus. It develops into one or more watery, painful blisters in the next few days. There can be burning or pain on urination, with swollen lymph nodes in the groin. There is an increase in discharge, or a feeling of pressure in the pelvic area. In some cases, the entire body reacts with flu-like symptoms: fever, headache, and chills.

Ninety percent of women develop sores on the vagina and cervix during a first infection. The blisters burst quickly, and shed highly contagious viruses everywhere. The now-empty blisters turn into shallow ulcers, which can be painful. The ulcers form into crusts, which heal spontaneously within 1 to 5 weeks. Visit the physician as soon as the symptoms appear. At an early stage, diagnosis can be made by sight alone. Help can begin immediately, but a culture test is very expensive.

At least 5 types of herpes virus are known to affect humans. The Epstein Barr virus and-cytomegalovirus causes infectious mononucleosis, also known as glandular fever. The varicella virus causes chicken pox in children, and shingles in adults. There are 2 types of herpes simplex virus. HSV 1 causes cold sores on the lips or nose, also called fever blisters. HSV 2 causes genital ulcers, also called genital herpes.

By adulthood, most people have been infected with the cold sore virus, HSV 1. They develop antibodies against it, and only a few actually get cold sores. Fewer adults have HSV 2 antibodies because the virus is spread by sexual contact. The findings of a recent study suggest that 99 percent of prostitutes have HSV 2 antibodies in their blood, compared with 3 percent of nuns and 29 percent of women in a committed relationship.

About 50 percent of those with HSV 2 have no symptoms. The recent increase in genital herpes is thought to be partly due to this, and partly due to an increase in the practice of oro-genital sex. In some cases, both HSV 1 and HSV 2 cause genital herpes. If suffering from a cold sore, avoid kissing, and any facial or genital contact. This applies to a partner as well.

Not all HSV 2 die after a first attack. The virus coats itself in the person’s own protein substance and retreats along nerve endings to the base of the spine. Here it sets up a permanent home, staying inactive for varying lengths of time. When the virus becomes active again, it usually returns to the same place as the previous attack. Recurring outbreaks can be virulent and painful, or very mild. If mild, a woman may be unaware that she is shedding highly contagious germs.

HSV 2 is particularly dangerous for women. It is linked with cancer of the cervix, The virus can cause miscarriage in the first 3 months of pregnancy. If shed during birth, 1 in 2 babies will be infected. Two out of 3 of those infected babies will die. Half the others suffer brain damage, or visual defects. These horrors are now avoided by Caesarian birth. The baby is lifted from the uterus and thus avoids contact with the virus.

As yet, there is no drug to destroy the herpes virus. The drug acyclovir helps reduce the pain of an attack; it may even lessen the number of recurrences. One of the miserable factors of herpes is the permanent risk of passing on the disease. Some physicians believe that this is only during the active phase; others strongly disagree. An infected person cannot be free of this worry.

Genital Warts

Molluscum Contagiosum: There are two kinds of warts, simple and genital. Both can infect the genitals; it is crucial to recognize the difference. Simple warts are the kind which appears on the hands of children. They are small, dimpled papules, which look like spots with a drop of pearly fluid inside. They are highly contagious, as their Latin name shows. They can be transmitted to the genitals by self or partner from warts on the hands and elsewhere. The virus enters the skin through invisible lesions which occur during sexual activity. The warts appear some 30 days after contact. Attacks of simple warts on the genitals are rare, being most likely in the teens and 20 to 30 age group.

If the penis is infected with simple warts, some men try self therapy. This is not advisable for women. Simple warts can be painful if rubbed, otherwise a woman is unaware of them. They are not life-threatening, nor do untold damage, but they are highly contagious. Visit the physician or clinic. Therapy varies.

Human Papilloma Virus: HPV is specific to the genital area. It is transmitted by direct sexual contact. The warts appear 3 weeks to 3 months after contact, but the incubation time can be up to 8 months, even more. The warts can be single; usually, they grow in clusters like grapes. With their raised, bumpy tops, they look like miniature cauliflowers. They grow on the labia lips or anus, inside the vagina, or on the cervix. In many cases, they are asymptomatic, and the woman is unaware that she is infected.

The warts are painless, but easily irritated by rubbing, and sometimes they itch. If there has been anal contact, they can grow inside the rectum and around the anus. More rarely with oral contact, they infect the linings of the mouth. If the warts breed in colonies on the cervix, the disease may not be detected until a Pap smear is done. Women with HPV have a five times higher risk of cancer of the cervix.

Larger warts, especially on the cervix, maybe vaporized by laser therapy, but it is difficult to know if they have all been destroyed. The healing process takes 6 weeks. Repeat therapy is necessary if they flare up again; avoid losing patience as laser therapy usually works. Other therapies include burning the warts off by electric cautery, or freezing them with dry ice. The physician then snips them off. External warts can be painted with the drug podophyllin. It takes 3 or 4 weekly treatments for the warts to dry up and drop off.

HPV infection is also called condyloma. The prescription drug Condylox has just been made available for home treatment, which means that patients no longer need to have a physician apply the therapy. At least 56 different types of the virus have been identified.

Hepatitis

Hepatitis A and B are caused by virus infection of the liver. The virus breeds in waste matter from the bowel and is common where there is poor sanitation. It is passed in contaminated food and drink; less usually, by sexual contact; more rarely, by transfusions of infected blood. Hepatitis is on the increase, probably due to more foreign travel. When visiting areas with poor sanitation, observe strict personal hygiene. Drink bottled water, eschew ice cubes. Avoid anal and oral sexual contact.

The symptoms of both A and B are the same: fever, nausea, headache, fatigue, loss of appetite, and chills. Jaundice shows as a yellow tinge to the skin, fingernails, and whites of the eyes about a week later. Urine can be dark in colour; stools almost whitish. A few people are asymptomatic. With hepatitis A, the symptoms are mild. The defence system builds immunity to the virus, but it remains in the blood and can be transmitted.

The hepatitis B virus (HBV) produces severe symptoms, which start suddenly 1 to 6 months after contact. If liver damage is extensive, death occurs in 5 to 20 percent of cases. The B virus is transmitted in blood and blood products during sexual contact: semen, vagina secretions, saliva, and faeces are suspect. It is also passed by IV drug users sharing infected needles. The incidence of HBV is rising rapidly, perhaps due to more foreign travel and IV drug use. Male homosexuals, heterosexuals with multiple partners, travellers, and drug addicts are high risk groups.

AIDS

AIDS stands for Acquired Immune Deficiency Syndrome.

Acquired: it is passed on, but not inherited.
Immunodeficiency: the immune system grows weak and deficient.
Syndrome: a group of symptoms of which the cause is unknown.

However, it is now known that AIDS is caused by the human immunodeficiency virus (HIV). The word AIDS is still used to avoid confusion. The virus does not kill, but it damages the immune system, leaving the person vulnerable to rare infections and cancers which are life-threatening. If death occurs, it is not from AIDS, but from one of these opportunistic diseases.

HIV is transmitted in body fluids: blood, blood products, semen, vagina secretions, and breast milk. It does not appear to be easily transmitted in saliva.

The Future

Scientists using an experimental AIDS vaccine have succeeded in changing the way the body fights the AIDS virus. The discovery could open the door to new ways of treating the disease. By giving the vaccine to 30 men and women infected with HIV, researchers found that they were able to prompt the immune systems of most in the group into mounting a more sophisticated counterattack against the virus. It is too early to know if this response will help HIV-infected people to survive the ravages of the disease.

The study’s results counter the long-standing and pessimistic conviction of many AIDS researchers that there is little to be done to improve upon the immune system’s battle against the HIV virus.

New therapies such as the use of the antiviral drug AZT early in infection and inhaled pentamidine to prevent AIDS-caused pneumonia will delay the time when HIV infection develops into full-blown AIDS.

Categories
Men's Health

Sexual Function

To establish at least a minimum of patient screening, at the onset of the clinical treatment program, no units were accepted in therapy unless the complaining partner in the couple (e.g., the impotent male or the non-orgasmic female) had a history of at least six months of prior psychotherapeutic failure to remove the symptoms of sexual dysfunction. Very soon this proved to be a poorly contrived standard, of little screening value.

As should have been apparent at the onset, there was no secure way of establishing the functional effectiveness of the prior therapeutic program. How determined and well oriented was the therapist, how cooperative or fully responsible was the patient? After two years this original standard was abandoned in favor of that currently in effect.

Sexual Screening

A reasonably effective method of screening has been substituted by requiring that no patients be accepted at the Foundation unless they have been referred by the authority. As the authority, the Foundation accepts physicians, psychologists, social workers, and theologians.

Beyond screening the patients for appropriate referral to the Foundation, the referral source further is asked to provide available details of psychosocial background relevant to the husband and wife’s sexual dysfunction.

A telephoned report is made to the referring authority describing husband and wife’s progress (or lack of it) during or immediately following the acute phase of treatment at the Foundation. The well-informed authority then can provide the most important reinforcement for newly acquired patterns of sexual interaction for the couple once removed from the Foundation’s direct control by the termination of the acute phase of therapy.

In many instances, patients in established psychotherapeutic programs have been referred for removal of symptoms reflecting a somewhat broad area of distress in which sexual inadequacy is only a part. After their two weeks at the Foundation, these couples are, of course, returned to referring authority to continue their established treatment programs.

Obviously, the referring authority, before continuing in therapy with his patient, is briefed in detail as to the couple’s response to its Foundation exposure. The screening process as currently constituted has several aims, all obviously selective in nature.

Symptoms of Sexual Inadequacy

Primarily, control which prevents referral of major psychopathology is presumed. In other words, the psychoneurotic is acceptable, but not psychotic.

It should be emphasized that the reversal of symptoms of sexual inadequacy in psychoneurotic patients is indeed a significant portion of the Foundation’s objectives. Acceptance of this role by the Foundation is based on the premise that the reversal of particularly troublesome sexual symptoms may speed the progress of a psychoneurotic patient within the greater context of his established and broader-based psychotherapy.

However, the majority of the couples contending with sexual dysfunction do not evidence psychiatric problems other than the specific symptoms of sexual dysfunction. Socio-cultural deprivation and ignorance of sexual physiology, rather than psychiatric or medical illness, constitute the etiologic background for most sexual dysfunction.

Therefore, when a couple is properly educated in sexual matters, and their specific symptoms are reversed, there is no need for further psychotherapy unless the extensive duration of the distress has created psychosocial complications no longer directly related to the sexual dysfunction.

Other areas of selective screening for information vital to the therapeutic program center on such questions as:

  1. Are both members really interested in reversing their basic dysfunctional status? If one member of the unit simply has no interest whatsoever in reversing the symptomatology of sexual dysfunction in the marital relationship, the unit probably needs legal rather than medical or behavioral advice. The chances of reversing sexual dysfunction under the circumstances of total disaffection for a marital partner are negligible.
  2. What, if anything, is known of the couple’s adjustment or maladjustment to its social community?
  3. Do the referred members of the couple understand the programs, procedures, and policies of the Foundation? If not, it is suggested that the local authority, quietly briefed in advance by the Foundation’s professional staff, present the information in more specific detail to his patients.
  4. What is the couple’s basic financial picture? Should the Foundation offer the patients an adjusted fee scale or free care?

Sexual Therapy Commitment

The original research premise emphasized the fact that the positive reversal of symptoms of sexual inadequacy during the acute phase of the treatment program was not of great importance. If there were to be any clinical claim for positive effect in the Foundation’s concentrated approach to symptom reversal, the clinical results would have to be judged in retrospect over a significant period of time, not at the termination of the acute phase of therapy.

Therefore, the policy of five years of follow-up for couples after the termination of the rapid-treatment phase of the program became an integral part of research standards. Failures to reverse symptoms are, of course, considered most significant.

Little clinical value can be established for any therapeutic program, regardless of the length of its ongoing treatment phase, if the results are not evaluated in long-term follow-up after the termination of the acute phase of therapy. The abiding guide to treatment value must not be how well the patients do under authoritative control but how well they do when returned to their own cognizance without therapeutic control.

This result finally must place the mark of clinical failure or success upon the total therapeutic venture.

Individual members of couples seen in treatment must agree to cooperate with five years of follow-up after the termination of the acute phase of the therapy program. They fully understand.

The Foundation’s basic premise that success in a reversal of the symptoms of sexual dysfunction means little during the two weeks of intensive treatment unless the symptom reversal is maintained for at least the first five years after separation from direct Foundation influence.

Success in the maintenance of symptom reversal for this length of time does provide some sense of permanency in the continuing effectiveness of the couple’s sexual functioning.

Those couples whose acute treatment phase was judged inadequate or a failure arbitrarily have not been placed in the five-year follow-up program. This type of follow-up would indeed have been a study of major importance, but such continuing interrogation certainly could have interfered seriously with other clinical approaches designed to relieve the unit’s problems of sexual dysfunction.

The therapy concepts and clinical procedures depict the basic methodology of co-therapist interaction, first, between team members, and second, directed toward husband and wife of the sexually dysfunctional marital, unit. Jules Masserman has so aptly described psychotherapy as “anything that works.” This “works” in a healthy percentage of cases.

Categories
Men's Health

Male Impotence Causes

A typical history of an acute episode of alcohol consumption as an etiological factor in the onset of secondary impotence is classic in its structural content. The clinical picture is one of acute psychic trauma on a circumstantial basis, rather than the chronic psychosocial strain of years of steady attrition to the male ego as described in the case history for the premature ejaculator.

There has been a specific history of onset of symptoms of secondary impotence as a direct result of episodes of acute alcoholic intake in 35 men from a total of 213 men referred with a complaint of secondary impotence.

The onset of secondary impotence in an acute alcoholic episode is so well known that it almost beggars description. A composite example is that of a relatively “successful” male aged 35-55, college graduate, working in an area which gears productive demand more to mental than physical effort.

The perfect environmental situation for onset of secondary impotence is any occupational hazard where demands for high levels of psychosocial performance are irrevocably a part of the nine-to-five day and frequently carry over into an evening of professional socializing.

Alcohol Impotence

Mr. A is a man with a habit of alcohol before dinner, frequently a few glasses of wine with his meals, and possibly a whisky. Alcohol intake at lunch is an integral part of his business as well.

In short, consumption of alcohol has become a part of his life.

This man and his wife leave home one night for a party and alcohol is available in large quantity. Somewhere in the late evening, the party comes to an end. Mr. A has had entirely becomes tipsy and so his wife drives them home for safety’s consideration.

His wife retires to the bedroom, and with a sense of vague irritation, a combination of a sense of personal rejection and a residual of her social embarrassment, prepares for bed. Mr. A has stumble but with the aid of a strong banister and even stronger nightcap, manages to arrive at the bedroom door. Suddenly he felt that his wife is indeed fortunate tonight, for he is prepared sexually satisfied her.

Alcohol Hangover

It never occurs to him that all she wants to do is go to bed and avoid a quarrel at all costs. He jumped into the bed, moves to meet his imagined commitment, and nothing happens. He has simply had too much alcohol.

Dismayed and confused both by the fact that no erection develops and that his wife obviously has little or no interest in his gratuitous sexual contribution, he pauses to resolve this complex problem and immediately falls into deep, anesthetized slumber.

Next day, he is further traumatized by the symptoms of an acute hangover. He surfaces later in the day with the concept that things are not as they should be. The climate seems rather cool around the house. He can remember little of the prior evening’s festivities except his deeply imbedded conviction that things did not go well in the bedroom. He is not sure that all was bad but he also is quite convinced that all was not good.

Obviously he cannot discuss his problem with his wife, she probably would not speak to him at this time. So he putters and mutters throughout the evening and goes to bed early to escape. He sleeps restlessly only to face the new day with a vague sense of alarm, a passing sense of frustration, and a sure sense that all is not well in the household this Monday morning.

He pondered about it over a drink or two at lunch and another, and while contending with traffic on the way home from work, decides to check out this evening the little matter of sexual dysfunction, which he may or may not have imagined.

Sexual dysfunction within 48 hours!

If the history of this reaction sequence is taken accurately, it will be established that Mr. A does not check out the problem of sexual dysfunction within 48 hours of onset, as he had decided to do on his way home from work. He arrives home, finds the atmosphere still markedly frigid, makes more than his usual show of affection to the children, retires to the security of the cocktail hour, and goes to supper and to bed totally lacking in any communicative approach to his frustrated, irritated marital partner.

Tuesday morning, while brushing his teeth, Mr. A has a flash of concern about what may have gone wrong with his sexual functioning after the party night. He decides unequivocally to check the situation out tonight.

Instead of thinking of the problem occasionally, his concern for “checking this out” becomes of paramount importance. On the way to work and during the day, he does not think about what really did go wrong sexually because he does not know. Rather he worries constantly about what could have gone wrong.

Needless to say, there is resurgence of concern for sexual performance during the afternoon hours, regardless of how busy his schedule is.

Mr. A leaves the office in relatively good spirits, but thoroughly aware that “tonight’s the night.” He does have vague levels of concern, which suggest that a little relaxation is in order; so he stops at his favorite tavern for a couple of drinks and arrives home with a rosy glow to find not only a forgiving, but an anticipatory, wife, ready for the reestablishment of both verbal and sexual communication that a drink ‘or two together before dinner can bring.

Probably for the first time in his life, he approaches his bedroom on Tuesday night in a self-conscious “I’ll show her” attitude. Again there has been a little too much to drink-not as much as on Saturday night, but still a little too much.

And, of course, he does show her. He is so consumed with his conscious concern for effective sexual function (the onset of his fears of performance) that, aided by the depressant effect of a modest level of alcoholic intake (modest by his standards), he simply cannot “get the job done.”

When there is little or no immediate erective reaction during the usual sexual preliminaries, he tries desperately to force the situation-in turn, anticipating an erection, then wildly conscious of its abscence, and finally demanding that it occur. He is consciously trying to will sexual success, while subjectively watching for tumescence. So, of course, no erection.

While in an immediate state of panic, as lie sweats and strains for the weaponry of male sexual functioning, he simultaneously must contend with the added distraction of a frightened wife trying to console him in his failure and to assure him that the next night will be better for both of them.

Sexual Incompetence

Both approaches are equally traumatic from his point of view. He hates both her sympathy and blind support which only serve to underscore his “failure,” and reads into his wife’s assurances that probably he can do better “tomorrow” a suggestion that no longer can he be counted on to get the job done sexually when it matters “today.”

A horrible thought occurs to Mr. A. He may be developing some form of sexual incompetence. He has been faced with two examples of sexual dysfunction. He is not sure what happened the first time, but he is only too aware this night that nothing has happened. He has failed, miserably and completely, to conduct himself as a man.

He cannot attain or maintain an erection.

Further, Mr. A knows that his wife is equally distressed because she is frantically striving to gloss over this marital catastrophe. She has immediately cast herself in the role of the soothing, considerate partner who says, “Don’t worry dear, it could happen to anyone,” or “You’ve never done this before, so don’t worry about it, dear.”

In the small hours of the morning, physically exhausted and emotionally spent from contending with the emotional bath her husband’s sexual failure has occasioned, she changes her tune to “You’ve certainly been working too hard, you need a vacation,” or “How long has it been since you have had a physical checkup?” (Any of a hundred similar wifely remarks supposed to soothe, maintain, or support are interpreted by the panicked man as tacit admission of the tragedy they must face together: the progressive loss of his sexual functioning.)

From the moment of second erective failure,

72 hours after the first erection failure, this man may be impotent.

In no sense does this mean that in the future he will never achieve an erection quality sufficient for intromission.

Occasionally he may do so and most men do. It does mean, however, that any suggestion of wifely sexual demand either immediate in its specific physical intensity or pointing coyly to future sexual expectations may produce pressures of performances quite sufficient to reduce Mr. A to and maintain him in a totally no erective state.

In brief, fears of sexual performance have assumed full control of his psychosocial system.

Mr. A thinks about the situation constantly. He occasionally asks friends of similar age group how things are going, because, of course, any male so beleaguered with fears of sexual failure is infinitely desirous of blaming his lack of effective function on anything other than himself, and the aging process is a constantly available cultural scapegoat.

Sexual Approach

He finds himself in the position of the woman with a lifetime history of non orgasmic return who contends openly with concerns for the effectiveness of her own sexual performance and secretly faces the fear that in truth she is not a woman. In proper sequence he does as she has done so many times.

He develops ways and means to avoid sexual encounter.

He sits fascinated by a third-rate movie on television in order to avoid going to bed at the usual time with a wife who might possibly be interested in sexual expression. He fends off her sexual approaches and jumps at anything that avoids confrontation as a drowning man would at a straw.

His wife immediately notices his disinclination to meet the frequency of their semi established routine of sexual exposure. In due course she begins to wonder whether he has lost interest in her, if there is anyone else, or whether there is truth in his most recent assertion that he couldn’t care less about sex.

For reassurance that she is still physically attractive, the concerned wife begins to push for more frequent sexual encounters, the one approach that the self-pressured male dreads above all else.

Obviously, neither marital partner ever communicates his or her fears of performance or the depth of their concerns for the sexual dysfunction that has become of paramount importance in their lives. The subject either is not discussed, or, if mentioned even obliquely, is hastily buried in an avalanche of words or chilled by painfully obvious avoidance.

Sexual Anxiety

Within the next 3 months, Mr. A has to fail at erective attainment only another time or two before both husband and wife begin to panic.

She decides independently to avoid any continuity of sexual functioning, eliminate any expression of her sexual needs, and be available only should he express demand, because she also has developed fears of performance.

Her fears are not for herself, but for the effectiveness of her husband’s sexual functioning.

She goes to great lengths to negate anything that might be considered sexually stimulating, such as too-long kisses, handholding, body contact, caressing in any way. In so doing she makes each sexual encounter much more of a pressured performance and therefore, much less of a continuation of living sexually, but the thought never occur to her. All communication ceases.

Each individual keeps his own counsel or goes his own way. The mutual sexual stimulation in the continuity of physical exposure, in the simple physical touching, holding, or even verbalizing of affection, is almost totally withdrawn.

The lack of communication that starts in the bedroom rapidly spreads through all facets of marital exchange: children, finances, social orientation, mothers-in-law, whatever.

In short :
Sexual dysfunction in the marital bed, created initially by an acute stage of alcoholic ingestion, supplemented at the next outing by ah “I’ll show her” attitude and possibly a little too much to drink can destroy the very foundation of a marriage of 10 to 30 years duration.

As the male panics, the wife only adds to his insecurity by her inappropriate verbalization, intended to support and comfort but interpreted by her emotionally unstable husband as immeasurably destructive in subjective content.

The dramatic onset of secondary impotence following an in stance of excessive alcohol intake is only another example of the human male’s extreme sensitivity to fears of sexual performance.

In this particular situation, of course, the onset of fears of performance was of brief but dynamic duration as opposed to those in the preceding example of the premature ejaculator whose fears of performance developed slowly, stimulated by continued exposure to his wife’s verbal denunciation of his sexual functioning.

Discussed above are examples of combinations of psychological and circumstantial factors that contribute the highest percentage of etiological input to the development of secondary impotence. Continuing through the listing of major influences there remain environmental, physiological, and iatrogenic factors.

In the final analysis:
Regardless of listing category, secondary impotence is triggered by combinations of these etiological factors rather than by any single category with the obvious exception of psychosocial influence. Once onset of erective failure has been recorded, regardless of trigger mechanism, involved, the individual male’s interpretation of or reaction to functional failure must be dealt with on a psychogenic basis.

The etiological factors recorded above are little more than categorical conveniences. From his initial heterosexual performance through the continuum of his sexual expression, every man constantly assumes a cultural challenge to his potency.

How he reacts to these challenges may be influenced directly by his psychosocial system, but of particular import is the individual susceptibility of the man involved to the specific pressures of the sexual challenge and to the influences of his background.

When considering etiological influences that may predispose toward impotence, it always should be borne in mind that most men exposed to parallel psychosexual pressures and similar environmental damage shrug off these handicaps and live as sexually functional males.

It is the factor of susceptibility to negative psychosocial input that determines the onset of impotence. These concepts apply to primarily as well as secondarily impotent men.

When considering environmental background as an etiological factor in secondary impotence, the home, the church, and the formative years are at center focus.

What factors in or out of the home during the formative years tend to initiate insecurity in male sexual functioning?

The preeminent factor in environmental background reflecting sexual insecurity is a dominant imbalance in parental relationships dominant, that is, as opposed to happen stance, farcical, or even fantasized battles for family control.

Secondary, but still of major import is the factor of homosexuality, which is to be considered in the environmental category. In no sense does this placement connote professional opinion that homophile orientation is considered purely environmental in origin.

Since homosexual activity may have derogatory influence upon the effectiveness of heterosexual functioning, the subject must be presented in the etiological discussion. The disassociations developing from homophile orientation are considered in the environmental category only for listing convenience.

Categories
Women's Health

Male Sex & Religion

While the multiplicity of etiological influences is acknowledged, the factor of religious orthodoxy remains of major import in primary orgasmic dysfunction as in almost every form of human sexual inadequacy.

Investigation of 193 women who have never achieved orgasmic return before referral to the Foundation for treatment, 42 were products of rigidly channelized religious control. Eighteen were from Catholic, 26 from Jewish, and 7 from fundamentalist Protestant backgrounds.

It may also be recalled that 9 of these 42 primarily non-orgasmic women reflecting orthodox religious backgrounds also were identified as having the clinical complaint of vaginismus, while 3 more women with orthodox religious backgrounds had to contend with situational orgasmic dysfunction and vaginismus simultaneously.

A history reflecting the control of orthodox religious demands upon the orgasmically dysfunctional woman and her husband is presented to underscore the Foundation’s professional concern for any orthodoxy-influenced imprinting and environmental input that can and does impose severely negative influences upon the susceptible woman’s psychosocial structure relative to her facility for sexual functioning.

Mr. A and His Wife

After 9 years of a marriage that had not been consummated, Mr. and Mrs. A were referred to the Foundation for treatment. He was 26 and she 24 years old at marriage. Mrs. A’s family background was one of unquestioned obedience to parents and disciplinary religious tenets.

She was one of three siblings, the middle child to an elder brother by three years, and a younger sister by two years. Other than her father, religion was the overwhelming influence in her life. The specific religious orientation that of Protestant fundamentalism encompassed total dedication to the concept that sex and sin were synonymous words.

Mrs. A remembers her father, who died when she was 19, as a Godlike figure whose opinion in all matters was an absolute law in the home. Control of dress, social commitment, educational direction, and in fact, school selection through college were his responsibility.

There were long daily sessions, of family prayer interspersed with paternal pronouncements, never family discussions. On Sunday the entire day was devoted to the church, with activities running the gamut of Sunday school, formal service, and young people’s groups.

The young woman described a cold, formal, controlled family environment in which there was complete demand for the dress as well as toilet privacy.

Not only were the elder brother and sisters socially isolated, but the sisters also were given separate rooms and encouraged to protect individual privacy.

She never remembers having seen her mother, father, brother, or sister in an undressed state. The subject of sex was never mentioned, and all literature, including newspapers, available to the family group was evaluated by her father for possibly suggestive or controversial material. There was a restricted list of radio programs to which the children could listen.

Mrs. A had no concept of her mother except as a woman living a life of rigid emotional control, essentially without a described personality, fully dedicated to the concept that a woman’s role was one of service. She considered it her duty and her privilege to clean, cook, and care for children, and to wait upon her husband.

There is no recall of pleasant moments of quiet exchange between mother and daughter, or, for that matter, of any freedom to discuss matters of the moment with either her brother or her sister.

As a young girl, she was totally unprepared for the onset of menstruation. The first menstrual period occurred while she was in school she was terrified, ran home, and was received by a thoroughly embarrassed mother who coldly explained to the young girl that this was a woman’s lot.

She was told that as a woman she must expect to suffer this “curse” every month. Her mother warned her that once a month she would be quite ill with “bad pains” in her stomach and closed the discussion with the admonition that she was never to discuss the subject with anyone, particularly not with her younger sister. The admonition was obeyed to the letter.

The mother provided the protective materials necessary and left the girl to her own devices. There was no discussion of when or how to use the menstrual protection provided.

Menstrual cramping had its onset with the second menstrual period and continued to be a serious psychosocial handicap until Mrs. A was seen in therapy. She also described the fact that her younger sister was confined to bed with monthly frequency while maturing.

During the Teenage Years

Dating in groups was permitted by her father for church-social activities and occasionally, well-chaperoned school events. College, selected by her father, was a coeducational institution which was described by her as living by the “18-inch rule,” i.e., handholding was forbidden and 18 inches were required between male and female students at all times.

Her dating was rare and well chaperoned. After graduation, she worked as a secretary in a publishing house specializing in religious tracts. Here she met and married a man of almost identical religious background.

The courtship was completely circumspect from a physical point of view. The couple arrived at their wedding night with a history of having exchanged three chaste kisses, which not only was the total of their physical courtship but also represented the only times she remembered ever being kissed by a man. Her father had felt such a display of emotion unseemly.

The only time her mother ever discussed a sexual matter was the day of her wedding. Mrs. A was carefully instructed to remember that she now was committed to serve her husband. It would be her duty as a wife to allow her husband privileges.

The Husband Privileges

were never spelled out. She also was assured that she would be hurt by her husband, but that “it” would go away in time. Finally and most importantly, she was told that “good women” never expressed interest in the “thing.” Her reward for serving her husband would be, hopefully, in having children.

She remembers her wedding night as a long struggle devoted to divergent purposes. Her husband frantically sought to find the proper place to insert his penis, while she fought an equally determined battle with nightclothes and bedclothes to provide as complete a modest covering as possible for the awful experience.

The pain her mother had forecast developed as her husband valiantly strove for intromission.

Although initially there were almost nightly attempts to consummate the marriage, there was a total lack of success. It never occurred to Mrs. A that she might cooperate in any way with the insertive attempts.

And since this was to be her husband’s pleasure, it, therefore, was his responsibility.

She evidenced such a consistently painful response whenever penetration was attempted that frequency of coital attempt dwindled rapidly. The last three years before referral, attempts at consummation occurred approximately once every three to four months.

For 9 years this woman only knew that she was physically distressed whenever her husband approached her sexually and that for some reason the distress did not abate, Her husband occasionally ejaculated while attempting to penetrate, so she thought that he must be satisfied.

Whenever Mr. A renewed the struggle to consummate, she was convinced that he had little physical consideration for her. Her tense, frustrated, negative attitude, initially stimulated by both the pain and the “good woman” concept described by her mother, became in due course one of complete physical rejection of sexual functioning in general and of the man involved in particular.

When seen in therapy, Mrs. A had no concept of what the word masturbation meant. Her husband’s sexual release before marriage had been confined to occasional nocturnal emissions, but he did learn to masturbate after’ marriage and accomplished ejaculatory release approximately once a week, without his wife’s knowledge. There was no history of extramarital exposure.

Of interest is the fact that Mrs. A’s brother has been twice divorced, reportedly because he cannot function sexually, and her younger sister has never married. As would be expected, at physical examination Mrs. A demonstrated a severe degree of vaginismus in addition to the intact hymen.

In the process of explaining the syndrome of involuntary vaginal spasm to both husband and wife, the procedures described were followed in detail. When vaginismus was described and then directly demonstrated to both husband and wife.

It was the first time Mr. A had ever seen his wife unclothed and also the first time she had submitted to a medical examination.

There obviously were multiple etiological influences combining to create this orgasmic dysfunction, but the repression of all sexual material inherent in the described form of religious orthodoxy certainly was the major factor.

Under Foundation direction, the process of education had to include reorientation of both the sexual and social value systems. The influence of the psychosocial system was turned from a dominant-negative factor to a relatively neutral one during the acute phase of treatment.

This alteration in repressive quality allowed Mrs. A’s natural biophysical demand to function without determined opposition, and orgasmic expression was obtained. Obviously, the husband needed a definitive psychosexual evaluation as much as did his wife.

Categories
Women's Health

Male Sexual Dysfunction

Male Sexual Dysfunction

To be diagnosed as having primary orgasmic dysfunction, a woman must report a lack of orgasmic attainment during her entire lifespan. There is no definition of male sexual dysfunction that parallels this severity of exclusion.

A Male Is Judged Primarily Impotent:

The definition means simply that he has never been able to achieve intromission in either homosexual or heterosexual opportunity. However, he might, and usually does, masturbate with some regularity or enjoy occasions of partner manipulation to ejaculation.

For the primarily non-orgasmic woman, however, the definition demands a standard of total orgasmic responsivity.

The edict of lifetime non-orgasmic return in the Foundation’s definition of primary orgasmic dysfunction includes a history of consistent non-orgasmic response to all attempts at physical stimulation, such as masturbation, male or female manipulation, oral-genital contact, and vaginal or rectal intercourse.

In Short

Every possible physical approach to sexual stimulation initiated by self or received from any partner has been totally unsuccessful in developing an orgasmic experience for the particular woman diagnosed as primarily non-orgasmic.

If a woman is orgasmic in dreams or fantasy alone, she still would be considered primarily non-orgasmic.

Foundation personnel has encountered two women who provided a positive history of an occasional dream sequence with orgasmic return and a negative history of physically initiated orgasmic release.

However, no woman has been encountered to date that described the ability to fantasy to orgasm without providing a concomitant history of successful orgasmic return from a variety of physically stimulative measures.

There are salient truths about male and female sexual interaction that place the female in a relatively untenable position from equality of sexual response.

Of primary consideration is the fact of a woman’s physical necessity for an effectively functioning male sexual partner if she is to achieve a coitally experienced orgasmic return.

During coition, the non-orgasmic human female is immediately more disadvantaged than her sexually inadequate partner in that her performance fears are dual in character. Her primary fear is, of course, for her own inability to respond as a woman, but she frequently must contend with the secondary fear for the inadequacy of male sexual performance.

The outstanding example of such a situation is, of course, that of the woman married to a premature ejaculator. From mutual responsibility for sexual performance, the woman has only to make herself physically available to provide the male with ejaculatory satisfaction.

The premature ejaculator in turn makes himself available, there usually is little correlation between intromission, rapid ejaculation, and female orgasmic return during the episode.

Married Premature Ejaculator

The biophysically disadvantaged female usually is additionally disadvantaged from a psychosocial point of view. Not only is there the insufficient bio-physical opportunity to accomplish orgasmic return, but in short order, the wife develops the concept of being sexually used in the marriage.

She feels that her husband has no real interest in her personally nor any concept of responsibility to her as a sexual entity. Many times the wife might be at a peak of sexual excitation with intromission. Without fear for her husband’s sexual performance, she could be orgasmically responsive shortly after the coital connection, displaying a full bio-physical capacity for sexual response.

But as she sees and feels the male thrusting frantically for ejaculatory release, she immediately fears the loss of sexual opportunity, is distracted from the input of biophysical stimuli by that fear, and rapidly loses sexual interest.

With the negative psychosocial-system influence from the concept of being used more than counterbalancing the high level of biophysically oriented sexual tension she brought to the coital act, the orgasmic opportunity is lost.

A brief attempt should be made to highlight the direct association of male and female sexual dysfunction in marriage, for there were 223 couples referred to the Foundation for treatment with bilateral partner complaints of sexual inadequacy. By far the greatest instance of a combined diagnosis was that of a non-orgasmic woman married to a premature ejaculator.

Of the total 186 premature ejaculators treated in the 11-year program, 68 were married to women reported as primarily non-orgasmic and an additional 39 wives were diagnosed as situationally non-orgasmic. Thus, in 107 of the 223 marriages with bilateral partner complaints of sexual dysfunction, the specific male sexual inadequacy was premature ejaculation.

Since the in-depth descriptions of the premature ejaculator presented in the earlier topics include full descriptions of the problems of female sexual functioning in this situation, there is no need for a detailed history representative of the 68 women primarily non-orgasmic in marriages to prematurely ejaculating men.

Another salient feature in the human female’s disadvantaged role in coital connection is the centuries-old concept that it is a woman’s duty to satisfy her sexual partner. When the age-old demand for accommodation during coital connection dominates any woman’s responsivity, her own opportunities for orgasmic expression are lessened proportionately.

If a woman is to express her biophysical drive effectively, she must have the single-standard opportunity to think and feel sexually during coital connection that previous cultures have accorded the man.

The male

must consider the marital bed as not only his privilege but also a shared responsibility if his wife is to respond fully with him in coital expression. The heedless male driving for orgasm can carry along the woman already lost in high levels of sexual demand, but his chances of elevating to orgasm the woman who is trying to accommodate to the rhythm, depth, and power of his demanding pelvic thrusting are indeed poor.

It is extremely difficult to categorize female sexual dysfunction on a relatively secure etiological basis. There is such a multiplicity of influences within the biophysical and psychosocial systems that to isolate and underscore a single, major etiological factor in any particular situation is to invite later confrontation with pitfalls in therapeutic progression.

Categories
Women's Health

Male Libido

Random orgasmic inadequacy is illustrated in the history below. With but two episodes of orgasmic attainment in her life, Mrs. H provides a history of one manipulative and one coital effort to orgasmic release. Her two highlighted sexual experiences were as much of a surprise to her when they occurred as they were to her husband.

There seems to be a clinical entity of low sexual tension which by history does not represent specific trauma to a sexual or any other value system. If so, it is rare both in occurrence and in professional identification. Perhaps the case history reported below is representative of such a situation.

Mr. and Mrs. H

were referred to the Foundation after 11 years of marriage with the wife’s stated complaint that she was just not interested in sex. She was 47 and her husband 44 years old. Her childhood and adolescent years had been spent in comfortable surroundings. She was the eldest by three years of two sisters and reported a relatively uneventful, non-traumatic background for growth and development.

Mrs. H was a relatively attractive woman with a reasonable number of dating opportunities during her high school and college years. Despite thoroughly enjoying the social aspects of the dating opportunities, there was little sexual stimulation from the few petting experiences she accepted.

She never masturbated and recalled no awareness of pleasant pelvic sensations during her childhood.

Her mother was a relatively self-sufficient woman with multiple socio-cultural interests. She never discussed the material of sexual content with her daughter. When Mrs. H. was 15, her father was killed in an automobile accident.

After college, Mrs. H sought the opportunity for a professional career in the business world. She continued working throughout her twenties, doing exceptionally well professionally. There was established social opportunity, but she found herself resistant to both male and female (one occasion) approaches to the shared sexual experience.

Her resistance was not described as aversion. It was just that she was essentially unstimulated by any sexual approach and saw no point in a commitment without interest.

She had several women and men friends and many interests. She worked hard, enjoyed her vacations, traveled extensively, but simply avoided sexual approach. At age 36 she met and married a man three years her junior who was working in the same professional field. They formed their own business venture.

From Mrs. H’s point of view, the marriage was simply a form of a business merger. The same could not be said for her husband. He was very much interested in sexual functioning. He had been married for less than two years in his mid-twenties and listed a large number of sexual opportunities with a wide variety of experiences before this marriage.

Mrs. H was totally cooperative in sexual functioning but was basically unmoved. She lubricated well with coital connection, found pleasure in providing a release for her husband, but was totally uninvolved personally.

She had never masturbated, and her husband’s attempts to stimulate her not only were unsuccessful but at times she even found them amusing when “nothing happened.” Neither repulsed nor frustrated, she simply wasn’t involved in sexual expression.

This was not her husband’s reaction to their mutual sexual experiences. He found her lack of responsiveness utterly frustrating. Together they prospered from a financial point of view, but her obvious lack of sexual interest was depressing to him as an individual:

Eighteen months before referral to the Foundation, Mrs. H was highly stimulated on one occasion during coital connection and was orgasmic. The couple thought success had been attained, but subsequent coital episodes found her essentially unstimulated. There was one other such episode of orgasmic attainment.

On this occasion, the business had gained an important new source of financial return and the unit had celebrated its success with dinner and the theater. She was orgasmic that night by manipulation only. Thereafter, there was no significant level of response regardless of the mode of stimulation. It was a high level of male frustration that brought the unit to the Foundation for treatment. Through the above article, we can recommend you the latest dresses.in a variety of lengths, colors and styles for every occasion from your favorite brands.

Orgasm and Masturbation

These were a few cases of masturbatory orgasmic inadequacy. The classification represents a stage of a woman’s sexual responsivity and, other than for categorizing purposes has no assigned value and will not be illustrated in-depth. Two types of history dominate this classification.

The first: is the story so often obtained from women guilt-ridden from masturbatory experimentation. They try to masturbate as young women, and after failing a time or two, simply withdraw from experimentation with the concept that they have fallen from grace. Later in their mature sexual experience, genital-area manipulation as a means of sexual excitation is at best moderately successful, but they are not orgasmic except during coition.

The second: is that of the female “don’t touch” syndrome. When taught that masturbation is evil they react by avoiding any approach to self-stimulation during adolescence and their maturing years. They may be orgasmic during socially acceptable coital opportunities but cannot be manually or orally elevated to orgasmic return.

The sexually dysfunctional woman as an effect of the male sexual function has been discussed in depth. There are so many variations on the theme of orgasmic inadequacy that many chapters could have been written, and the subject still would not have been covered adequately.

The concepts of a duality of psychosocial and biophysical structuring that influence a woman’s sexual response patterns have been advanced. If any woman’s sexual value system is either undeveloped or damaged by an imbalance of either of these two theoretical systems of influence, the return may be varying degrees of orgasmic inadequacy.

When faced with the clinical responsibility of treatment demand for primary or situational orgasmic dysfunction, the therapist must have established theoretical concepts of sexual dysfunction if he is to treat effectively.

Categories
Women's Health

Male Female Sexual Response

Male Female Sexual Response

Both contribute positively or negatively to any state of sexual responsivity but have no biological demand to function in a complementary manner.

With the reminder that finite analysis of male sexual capacity and physiological response also has attracted little scientific interest in the past.

Compare Male And Female

It should be reemphasized that similarities rather than differences are frequently more significant in comparing male and female sexual responses. By intent, the focus of this topic is directed toward the human female, but much of what is to be said can and does apply to the human male.

The bio-physically and psychosocially based systems of influence that naturally coexist in any woman have the capacity if not the biological demand to function in mutual support.

Obviously, there is an interdigitation of systems that reinforce the natural facility of each to function effectively. However, there is no factor of human survival or internal biological need defined for the female that is totally dependent upon a complementary interaction of these two systems.

Unfortunately, they frequently compete for dominance in problems of sexual dysfunction.

Woman’s Response

When the human female is exposed to negative influences under circumstances of individual susceptibility, she is vulnerable to any form of psychosocial or biophysical conditioning, i.e., the formation of man’s individually unique sexual value systems.

Based on how an individual woman internalizes the prevailing psychosocial influence, her sexual value system may or may not reinforce her natural capacity to function sexually.

One need only remember that sexual function can be displaced from its natural context temporarily or even for a lifetime to realize the concept’s import.

Women cannot erase their psychosocial sexuality and sexual identity, being female, but they can deny their biophysical capacity for natural sexual functioning by conditioned or deliberately controlled physical or psychological withdrawal from sexual exposure.

Yet a woman’s conscious denial of biophysical capacity rarely is a completely successful venture, for her physiological capacity for sexual response infinitely surpasses that of man.

Indeed, her significantly greater susceptibility to negatively based psychosocial influences may imply the existence of a natural state of psycho-sexual-social balance between the sexes that has been culturally established to neutralize a woman’s biophysical superiority.

The specifics of the human female’s physiological reactions to effective levels of sexual tension have been described in detail, but brief clinical consideration of these reactive principles is in order.

For women, as for men, the 3 specific total-body responses to elevated levels of sexual tension are:

  1. Increased myotonia or muscle tension
  2. Generalized vasocongestion, pooling of blood in tissues
  3. Sex flush and breast enlargement.

When clinical attention is directed toward female orgasmic dysfunction, one particular biological area, the pelvic structures is of the moment.

Specific evidence has been accumulated from the incidence of both myotonia and vasocongestion in the female’s pelvis.

Categories
Women's Health

Inexperience Sexual Male

For many women, one of the most frequent causes for orgasmic dysfunction, either primary or situational, is a lack of complete identification with the marital partner.

The husband may not meet her expectations as a provider. He may have physical or behavioral patterns that antagonize.

Most Important

He may stand in the place of the man who had been much preferred as a marital partner but was not available or did not choose to marry the distressed woman. For myriad reasons, if the husband is considered inadequate according to his wife’s expectations, a negative dominance will be created in the psychosocial structure of many women.

Such a situation is exemplified by the following:

Mr. and Mrs. C

were 46 and 42 years of age, respectively, when referred to the Foundation. The wife complained of a lack of orgasmic return. The couple had been married 19 years when seen in treatment. The marriage was the only one for either partner. There were three children, the eldest of whom was 17, the youngest 12. There were barely adequate financial circumstances.

Mrs. C’s adolescent background had been somewhat restrictive. Her mother was a dominant woman with whom she developed little rapport. Her father died when she was 9 years old. There was one other sibling, a sister 8 years younger. Mrs. C went through the usual high school preparation, had two years of college, and then withdrew to take secretarial training and go to work in a large manufacturing company.

During her formative years, there were several friends, none of them particularly close except for one girl with whom she shared all her confidences. Mrs. C as a girl was fairly popular with boys, dated with regularity, and went through the usual petting experiences, but decided to avoid coital connection until marriage. She had no masturbatory history but described pleasure in the petting experiences, although she was not orgasmic.

Shortly after her twenty-second birthday, she fell in love with a young salesman for the company in which she worked. Theirs was a very happy relationship with every evidence of real mutuality of interest. She came to know and thoroughly enjoy his family, and they made plans to marry.

Three weeks before the marriage, her fiance, on a business trip, met and a week later married another woman, a divorced with two children. The jilted girl was crushed by the turn of events. This had been her only serious romantic attachment, and it had been a total commitment on her part.

Their Sexual Expression: petting and manipulated her fiance to ejaculation regularly.

Although she had been highly stimulated by his approaches she had not been orgasmic. The coital connection had not been attempted.

Six months later she married Mr. C, whom she thought kind and considerate. Their sexual experiences together were pleasant, but she achieved nothing comparable to the high levels of excitation provided by the first man in her life.

She described life with her husband as originally a good marriage. The children arrived as planned and the husband continued to progress satisfactorily in his business ventures, but husband and wife had very few mutual interests.

As the years passed Mrs. C became obsessed with the fact that she had never been orgasmic. She began to masturbate and reached high levels of excitation. Straining and willing orgasmic return without being able to fully accept the unrealistic nature of her imagery and fantasying, she failed, of course, in accomplishment.

Inexperience Husband

Her husband, with very little personal sexual experience other than in his marriage, had no real concept of an effective sexual approach. She repeatedly tried to tell him of her need, but his cooperative effort, maintained for only brief periods of time, was essentially unsuccessful.

After 12 years of marriage, Mrs. C sought sexual release outside the marriage with a man sexually much more experienced than her husband.

He did excite her to high plateau levels of sexual demand, but she always failed to achieve orgasmic release. This connection lasted off and on for a year and was only the first of several such extramarital commitments, always with the same disappointment in sexual return.

She was never able to avoid the fantasy of her former fiance whenever she approached orgasmic return, but her fantasy included a primarily negative impetus. Her frustration at “marrying the wrong man” was a constant factor in her coital encounters, as it was in most other aspects of her life.

As time passed she blamed her husband increasingly for her lack of orgasmic facility and became progressively more discontented with her lot in the marriage. She began to find fault with his financial return and social connections.

In short, Mrs. C felt that her husband was not providing satisfactorily for her needs and inevitably compared him with the man “she almost married.” This man had become a relatively well-known figure in the local area, had done extremely well financially, and apparently had a happy, functioning marriage.

Although Mrs. C never saw her former fiance, she constantly dwelt on what might have been, to the detriment of the ongoing relationship. Mrs. C sought psychiatric support for her non-orgasmic status but was unable to achieve the only real goal in her life, orgasmic release.

Finally, the husband and wife were referred to the Foundation to overcome professionally the conditioning of an adult lifetime and to cope with the requirements of her sexual value system impaired by the trauma it sustained when she was jilted by a man with whom she identified totally.

It is necessary to adjust to both her social and her sexual value systems be made in the hope of reversing or at least neutralizing the negative input of her psychosocial structure. There is no possible means of restructuring the negative input from “I married the wrong man” unless the problem is attacked directly.

First, in private sessions, the immature deification of her former fiance must be underscored.

Second, Mr. C must be presented to his wife in a different light, not in a platitudinal manner, but as the female co-therapist objectively views him.

A man’s positive attributes as he appears in another woman’s eyes carry value to the dysfunctional woman. Then there must be stimulation of the biophysical structure to levels of positive input. This, of course, is initiated by sensate-focus procedures.

Finally, the contrived somatic stimulation must be interpreted to Mrs. C’s sexual value system both by the co-therapists and by her husband. If these treatment concepts are followed successfully there is every good chance to reach the goal of orgasmic attainment.