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Sexual Dysfunction Treatment

Sexual Dysfunction Treatment

In any approach to a psycho-physiological process, treatment concepts vary measurably from school to school and, similarly, from individual therapist to individual therapist. The Reproductive Biology Research Foundation’s theoretical approaches to the treatment of men and women distressed by some form of sexual dysfunction have altered significantly and, hopefully, have matured measurably during the past 11 years. There are founded on a combination of 15 years of laboratory experimentation and 11 years of clinical trial and error.

Sexual Response

When the laboratory program for the investigation in human sexual functioning was designed in 1954, permission to constitute the program was granted upon a research premise which stated categorically that the greatest handicap to successful treatment of sexual inadequacy was a lack of reliable physiological information in the area of human sexual response.

It was presumed that definitive laboratory effort would develop material of clinical consequence. This material in turn could be used by professionals in the field to improve methodology of therapeutic approach to sexual inadequacy. On this premise, a clinic for the treatment of human sexual dysfunction was established at Washington University School of Medicine in 1959, approximately five years after the physiological investigation was begun. The clinical treatment program was transferred to the Reproductive Biology Research Foundation in 1964.

When any new area of clinical investigation is constituted, standards must be devised in the hope of establishing some means of control over clinical experimentation. And so it was with the new program designed to treat sexual dysfunction. Supported by almost five years of prior laboratory investigation, fundamental clinical principles were established at the onset of the therapeutic program. The original treatment concepts still exist, even more strongly constituted today. As expected, there were obvious theoretical misconceptions in some areas, so alterations in Foundation’s policy inevitably have developed with experience.

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Sexual Dysfunction

Orgasmic Dysfunction

The potential for orgasmic dysfunction: highlighted in the psychosocial-sexual histories of those women in marital units referred to the Foundation can be described in composite profile.

A baseline of dysfunctional distress was provided by specific material recalled not only from sexually developmental years but further encompassing all opportunities of potential sexual imprinting, conditioning, and experience storage.

Described in many settings, the dissimulation of sexual feeling consistently was reported as a manifest requirement or as a residual of earlier learning, operant as a requirement. Imprinting is that process which helps define the behavioral patterns of sexual expression and signal their arousal.

Dysfunction Origin
of the negative conditioning varied widely. At one pole it represented the influence of deliberate parental omission of reference to or discussion of sexual function as a component of the pattern of living. This informationally underprivileged background also failed to provide any example of female sexuality, recognizably secure in expression, which could be emulated.

In both situations, the sexually and socially maturing young woman was left to draw formative conclusions by negative implication, or, in the absence of this form of direction, she was forced to react to any influence available from her socio cultural environment.

The other extreme of rejective conditioning was reported as rigidly explicit but consistently negative admonition by parental and/or religious authority against personal admission or overt expression of sexual feeling.

Negative variants, there were many levels of uninformed guidance for the young girl or woman as she struggled with psychosocial enigmas, cultural restrictions, and her own physical sexual awareness.

Usually such guidance, though often well intentioned, was more a hindrance than a help as she developed her sexual value system and ultimately her natural sexual function.

In direct parallel to the degree to which the young girl developing a sexual value system seemed to have dissimulated her sexual interests during phases of imprinting, conditioning, and information storing, older women, now sexually dysfunctional, reported consistent precoital evidence of repression of sexual identity in mature sexual encounters.

Residual repression of sexual responsivity in the adult usually went well beyond any earlier theoretical requirements for a social adaptation necessary to maintain virginity, to restrain a partner’s sexual demand, or even to conduct interpersonal relationships in a manner considered appropriate by representative social authority. Not infrequently the residual repression of sexual responsivity was so acute as to be emphasized clinically with the time-worn cry.

Most primarily non orgasmic women
Repressed expression of sexual identity through ignorance, fear, or authoritative direction was the initial inhibiting influence in failure of sexual function.

Not infrequently this source of repression was identified as a crucial factor of influence for situationally non orgasmic women as well, although these individuals had the facility to overcome or circumnavigate its control under certain circumstances.

When requirements of the sexual value system prevailing during initial opportunities at sexual function could not be fulfilled because of the component of repression, each woman attempted without success to compensate in her desire for sexual expression by developing unrealistic partner identification, concept of social secureness, or pleasure in environmental circumstance.

Failure of her own sexual values to serve, there was almost a blind seeking for value substitutes. When a workable substitution was not identified and the void of psychosexual insecurity remained unfilled, sexual dysfunction became an ongoing way of life.

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Sex & Dyspareunia

Sex and Dyspareunia

The term dyspareunia, difficult or painful coitus, has always been presumed to refer to coital distress in women. The word stems from the Greek, and somewhat freely translates into “badly mated.”

Since no comparable word reflecting or suggesting coital distress for men has been established, poetic license will be begged. Here is comprised of two separate sections devoted to consideration of individual complaints of female and male sexual dysfunction identified by the individuals involved as difficult or painful coitus. Men can be “badly mated” too!

That factor in the total of male and female sexual dysfunction perhaps most difficult for the therapist to define involves the psycho physiological complaint of dyspareunia. Diagnostic insecurity relates directly to the fact that dyspareunia has a varied number of both subjective and objective origins that frequently give rise to combinations of psycho physiological distress rather than complaints that can be categorized individually.

Avoid Sex

For years, woman’s complaint sex hurts when had intercourse has been an anathema to the therapist. Even after an adequate pelvic examination, the therapist frequently cannot be sure whether the patient is complaining of definitive but undiagnosed pelvic pathology or whether, as has been true countless thousands of times.

A sexually dysfunctional woman is using the symptomatology of pain as a means of escaping completely or at least reducing markedly the number of unwelcome sexual encounters in her marriage.

For it is true that once convinced that there is no recourse for reversal of his or her dysfunctional status, the sexually inadequate partner in any marriage manufactures excuse after excuse to avoid sexual confrontation.

As women have long since learned, a persistent, aggressive male partner can overwhelm, neutralize, or even negate the most original of excuses to avoid sexual exposure.

However, presuming any degree of residual concern for or interest in his partner as an individual, the husband is rendered powerless to support his insistence upon continuity of sexual contact when the wife complains of severe distress during or after sexual connection.

If the female partner complains and flinches with penile insertion, moans and contracts her abdominal and pelvic musculature during the continuum of male thrusting, cries out or screams with deep vaginal penetration, sheds bitter tears after termination of every sexual connection, or complains angrily of aching in the pelvis or burning in the vagina during or even hours after a specific coital episode.

The male sexual approach must be accepted as the probable potentiator of a physiological basis for his female partner’s evidenced sexual dysfunction. Thereafter, the husband has minimal recourse. There is little he can do other than to avoid or at least reduce marital-unit sexual exposure on his own cognizance, and/or to insist that his wife seek professional consultation.

Once consulted, the twofold problem that constantly baffles authority is first whether a specific physiological basis can be defined for the objective existence of pain. Second, if not, whether the existence of pelvic pathology should arbitrarily be ruled out, thereby defining the registered complaint of dyspareunia as subjective in origin. When a woman complains of pain during or after intercourse, there are very few diagnostic landmarks to follow for treatment, so that consideration of the etiology of the painful response seems appropriate.

As in vaginismus, a differential diagnosis cannot be established for a complaint of dyspareunia unless careful pelvic and rectal examinations are conducted. Even then there can be no sure diagnosis if the existence of pelvic pathology is denied purely on the basis of negative examinations by competent authority.

Yet, in a positive vein, there are obvious pelvic or rectal findings that can and do support objectively a woman’s subjective complaint of coital discomfort. The female partner’s persistent complaint of pain with any form of coital connection must not be authoritatively denied or, for that matter supported, purely on the basis of interrogation, regardless of how carefully or in what depth the questioning has been conducted.

There are many varieties of dyspareunia, varying from postcoital vaginal irritation to severe immobilizing pain with penile thrusting. Symptomatic definition relating not only to the anatomy of the vaginal barrel but also to the total of the reproductive viscera is in order.

In no sense will the discussion include all possible forms of pelvic distress. Considered, however, will be the major sources of pelvic pathology engendering painful response from the female partner during or after coital connection. The dyspareunia will be considered in relation to specific areas of the vaginal barrel, the reproductive viscera, and the soft tissue components of the pelvis, and to painful stimuli developing, in a time-related sequence during or after coital connection.

Sex and Painful Vagina

The complaint of pain with penile intromission should demand clinical inspection of the vaginal outlet and the labial (major and minor) area. Direct observation can easily delineate any of the following minor areas of concern, minor only in the sense of easy reversibility of physical distress by adequate clinical measures.

An intact hymen or the irritated or bruised remnants of the hymenal ring can and do cause outlet pain during attempted coital connection. Less obvious is an unprotected scar area just at the mucocutaneous juncture of the vaginal mucosa and the perineal body.

These scars, primarily residuals of episiotomies sustained during childbirth, occasionally have been observed to result from criminal abortion techniques or gang-rape episodes. The Bartholin-gland area in the minor labia should be carefully palpated for enlargement in the gland base, which can contribute to a locally painful reaction as the vaginal outlet is dilated by the penile glans at onset of intromission.

Finally, in postmenopausal women the labia and vaginal outlet may have so lost elasticity and become so shrunken in size that any penile insertive attempt will return a painful response.

Sex and Clitoris Irritation

With any complaint of outlet pain, the clitoral area also should be inspected carefully. Many women simply cannot define anatomically or are too embarrassed to discuss objectively the exact location of the outlet distress occasioned by attempts at coital connection.

Smegma beneath the clitoral foreskin can cause chronic irritation and burning that becomes severe as the penis is introduced into the vaginal orifice. Rarely adhesions beneath the minor labial foreskin anchoring the foreskin to-the clitoral glans can cause distress when the foreskin is moved or pulled from its specific pudendal-overhang position by manipulative approaches to the mons area or by intromissive attempts.

When the minor labial hood of the clitoris is pulled down toward the perineum by the act of penile intromission, an intense pain response from the presensitized clitoral glans or even the clitoral shaft may become of major clinical moment.

The same type of reaction can be elicited if foreplay in the clitoral area has been irritative rather than stimulative in character, as so often happens when the sexually uneducated male tries to follow “authoritative” directions in attempts to stimulate his partner sexually. Heavy handed manipulation or frequent masturbatory irritation can elicit painful responses from the clitoral-glans area. This irritative reaction may develop rapidly

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Sex & Dyspareunia

Pelvic Disease

Endometriosis is a disease in which implants of endometrial tissue spread throughout the pelvic viscera and their protective covering, the peritoneum. When examined microscopically, this ectopic tissue resembles the lining of the inner cavity of the uterus.

The tubes, ovaries, broad ligaments, omentum, and the posterior wall of the uterus may be involved by firm fibrous adhesions. There are even many instances of tying together omentum and bowel with the reproductive viscera into large pelvic masses. The etiology of endometriosis has not been fully established.

It would not serve the purposes of this text to enter into a detailed discussion of the subject. Although endometrial implants appear in many anatomical areas other than the pelvic viscera, consideration will be focused alone on local pelvic implants.

Even if there are no major adhesions in the pelvis, there are at least minor elements of continuous local peritoneal irritation. Endometrial nodules usually can be felt most effectively with simultaneous manual pelvic-rectal examination.

The pain created by intercourse is due to the constriction and immobilization of the peritoneum and the firming up of the soft tissues of the pelvis by adhesions. The pelvic structures have progressively less facility to distend, expand, and move freely as the endometriosis progresses.

There is consequently more local tissue resistance to involuntary vaginal expansion, uterine elevation, and male pelvic thrusting.

In all situations that create chronic irritation of the pelvic peritoneum, fixation of the uterus, or constriction of the vaginal barrel, pain with intercourse is a relatively constant finding.

Treatment for endometriosis is either medical or surgical depending upon the degree of soft-tissue and pelvic visceral involvement. But once endometriosis has developed to a point at which there is significantly severe pain in response to coital activity, there must be definitive treatment of the condition, or the individual woman will have little hope of relief from the symptoms of progressively increasing dyspareunia.

Post Surgical

There are three important sources for acquired dyspareunia following removal of the uterus for specific organ pathology. First, dyspareunia results from thoughtless surgical technique. Physicians, when performing a hysterectomy, may overlook the fact that the cervix enters the vagina through the superior wall of that organ. When the wound in the vaginal barrel is repaired after removal of the cervix, if care is not taken to retain a superior position for the vaginal cuff, the scarred area, instead of being retained in the superior vaginal wall, may be pulled into the depth of the barrel by tissue constriction or by excessive folding or removal of vaginal tissue.

Postoperatively when the husband thrusts deeply into the vagina, the penis can come into contact with the resistant scarred area. There is little residual facility for involuntary vaginal distention in the area of the surgical scar.

Therefore, dyspareunia of significant proportion develops occasionally as a post surgical complication. Since this unfortunate result usually does not develop for months or even a year after surgery, the operating surgeon may never be made aware of the acquired dyspareunia.

The second opportunity to acquire dyspareunia is occasioned by the surgical indications for removal of the ovaries at the time the uterus is removed, or for that matter, at any time. If post operative sex-steroid-replacement is not initiated, many women will develop senile changes in the vagina and, in time, secondary dyspareunia.

The third incidence of dyspareunia after hysterectomy rarely comes to the attention of the operating surgeon. The etiology of the acquired dyspareunia may be subjective in origin.

Sexual Anxious Woman

If the woman facing hysterectomy and/or removal of the ovaries is not reassured with her husband that there need not be reduction of sexual drive or orgasmic facility after surgery, her fantasy and her friends’ old wives’ tales may, by power of suggestion, create fears of sexual performance for the anxious woman.

If she feels that she is going to be castrated, and sex-steroid-replacement therapy is not explained and offered as indicated, she well may believe that after surgery there will be loss of ability to respond in a sexually effective manner in the future. What is worse, an uninformed husband may have similar concepts.

If anything, sexual responsivity should be higher shortly after than immediately before surgery. The pelvic pathology for which the hysterectomy or oophorectomy is indicated usually detracts from sexual effectiveness by creating a state of ill health which, in turn, reduces innate sexual tension.

When the offending condition is removed and the general state of health consequently improved, there usually is a reawakening of sexual interest. If women are not reassured before surgery, many presume that, in the future, intercourse will provide no return for them or for their husbands, or that intercourse will even be painful.

Any woman has only to be sure that she will be distressed by future coital connection to take a long step toward acquired dyspareunia.

There are, of course, many factors other than the major ones of infection, endometriosis, post surgical objective and subjective complications, and the syndrome of broad-ligament laceration that create painful stimuli from irritated peritoneal and pelvic soft tissues in response to coital connection.

These include tumors of the uterus, such as myomas (fibroids), ovarian cysts and solid tumors, and, carcinoma of the female reproductive tract. Any of these tumor growths occasionally incite onset of the complaint of acquired dyspareunia. Those interested can find more definitive evaluations of this physiological source of dyspareunia in current gynecology textbooks.

Thus, the basic premise with which the Foundation approaches the problem of dyspareunia is one of elimination of possible pathological reasons for the complaint. If a woman complains of pain with intercourse, her complaint is accepted at face value, and steps are taken to identify the biophysical source of the coital distress.

The diagnosis of psychosomatic dyspareunia, unquestionably of moment in the sexual-response field, must be made by exclusion. To assign subjective origins to pelvic pain, regardless of the patient’s personality structure, without definitive physical evaluation of the pelvis, can result in clinical mismanagement of patients. Certainly there are times when, after every effort has been made to establish physical source of the pelvic pain, subjective etiology for the complaint will be considered strongly. But the initial bio physical investigative effort must be made by competent authority.

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Sex & Dyspareunia

Curve Penis

Peyronie
A disease produced by induration and fibrosis of the corpora cavernosa of the penis and evidenced as an upward bowing of the penis, plus a gradually increasing angulation to the right or left of the midline, makes coital connection somewhat difficult, and in advanced stages coition is virtually impossible.

There also may be pain attached to attempts at coital connection due to the unusual angulation of the penis creating resultant penile shaft strain, both with inserting and with thrusting experience.

Penile Chordee or Curved Penis

It is seen rarely in situations of penile trauma and only occasionally with neglected gonorrheal urethritis. Consultation has been requested by four men with severe penile chordee as a post traumatic residual.

In two instances the fully erect penis was struck sharply by an angry female partner. The remaining two men each described severe pain with a specific coital experience. During uninhibitedly responsive coital connection with the female partner in a superior position, the penis was lost to the vaginal barrel. In each case, the women tried to remount rapidly by sitting down firmly on the shaft of the penis.

The vaginal orifice was missed in the hurried insertive attempt and the full weight of the woman’s body sustained by the erect penis.

Each of the four men gave the remarkable verbal description that he felt or heard something snap. Shortly thereafter an obvious hematoma appeared on the anterior or posterior wall or lateral walls of the penile shaft.

Over a period of weeks, as the local hemorrhage was absorbed, fibrous adhesions developed and, with subsequent scar formation, there slowly developed a downward bowing and (in three cases) mild angulation of the penis.

Urologists state that due to the type of tissue involved in the penile trauma, there is little to offer in the way of clinical reprieve for men afflicted with these embarrassing erective angulations, Peyronie’s disease or chordee.

Attempts at surgical correction currently are of relatively little value and not infrequently make the situation worse. Any of these situations create responses of pain and tenderness during both masturbation and coital connection.

It always should be borne in mind that the erect penis can be traumatized by a sudden blow, by rapidly shifting coital position, by applying sudden angulation strain to the shaft, or from violent coital activity that places sudden weight or sudden pressure on the fully erect penis. The unfortunate residuals of such trauma have been described above.

Direct trauma of the penis occasioned by major accidents, war injuries, or direct physical attack sometimes requires that treatment for sexual dysfunction be patterned to include marked variation in the anatomical structuring of the penis. In anatomical deformity of the penis, the complaint of dyspareunia can be raised by either the male or female sexual partner.

Testicular Pain

Usually of the dull, aching variety, develops for some men who spend a significant amount of time in sexual play or in reading pornographic literature, concurrently maintaining erections for lengthy periods of time without ejaculating within the immediate present.

Frequent returns to excitement or even plateau-phase levels of sexual stimulation without ejaculatory relief of the accompanying testicular vasocongestion can cause an aching in either or both testes, particularly in younger men. Relief is immediate with ejaculation, which disperses the superficial and deep vasocongestion and returns the testicles to their normal size.

No permanent damage is occasioned by maintaining chronic testicular congestion for a period of days. Men with this syndrome of testicular pain occasioned by long-maintained sexual tension are in the minority.

Usually, the syndrome of involuntary testicular pain is relieved somewhat as the man ages.

There are painful reactions that develop during or shortly after coital connection that particularly reflect the influence of the vaginal environment. These situations are mentioned only in passing, but the therapist should keep in mind the fact that the basic pathology involved rests within the vaginal environment.

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Secondary Impotence

Secondary Impotence

Definition of secondary impotence depends upon acceptance of the concept of primary impotence as expressed and discussed in primary impotence. Primary impotence arbitrarily has been defined as the inability to achieve and/or maintain an erection quality sufficient to accomplish coital connection.

If erection is established and then lost from real or imagined distractions related to the coital opportunity, the erection usually is dissipated without an accompanying ejaculatory response. If diagnosed as primarily impotent, a man not only evidences erective inadequacy during his initial coital encounter but the dysfunction also is present with every subsequent opportunity.

If a man is to be judged secondarily impotent, there must be the clinical landmark of at least one instance of successful intromission, either during the initial coital opportunity or in a later episode. The usual pattern of the secondarily impotent male is success with the initial coital opportunity and continued effective performance with the first fifty, hundred, or even thousand or more coital encounters.

Finally, an episode of failure at effective coital connection is recorded.

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Secondary Impotence

Impotence Male

There are innumerable classic examples in the literature of maternal dominance contributing to secondary impotence. Thirteen such instances reflecting maternal dominance have been referred to the Foundation for therapy. Since the picture is so classic, a composite history can be provided to protect anonymity without destroying categorical effectiveness.

Impotence In Young Man

Maternal dominance primarily depreciates the young male’s security in his masculinity and destroys confidence in his socio cultural role-playing by eliminating or at least delimiting the possibility of a strong male image.

When the father is relegated to the role of second-class citizen within family structuring, the teenage boy has no male example with which to identify other than that of a devalued, shadowy, sometimes even ludicrous male allowed access to the home but obviously subject to control of the dominant maternal figure.

Mr. B, 34 years old
was referred with his wife for treatment of secondary impotence. He could remember little in family structuring other than a totally dominant mother making all decisions, large or small, controlling family pursestrings, and dictating, directing, and destroying his father with harsh sarcasm.

He remembered the paternal role only as that of an insufficient paycheck, and of a man sitting quietly in the corner of the living room reading the evening newspaper.

When he reached midteens, the parental representative at school functions was always the maternal figure, for both the young male and his younger sister (two siblings only). The same situation applied to church attendance and, eventually, to all social functions. The family matured with the concept that only three people mattered.

Masturbatory onset was in the early teens with a frequency of two or three times a week during the teenage years. As would be expected in a maternally dominated environment, dating opportunity for the boy was delayed, in this case until the senior year in high school.

Through college there were rare commitments to female interchange, all of them of a purely social vein. The young man was insecure in most social relationships, particularly those having orientation to the male sex.

He had been forbidden participation in athletics by his mother for fear of injury. He rarely pursued male companionship, feeling himself alternatively totally insecure in, or intellectually superior to, the male peer group.

Premarital sex in youth
Finishing college, the young man, particularly interested in actuarial work, joined an insurance firm. Although mainly withdrawn from social relationships, at age 28 he met and within three months married a 27 year old divorcee with a 2 year old daughter.

The divorcee, a dominant personality in her own right, was the mirror image of his mother. The two women were, of course, instant, bitter, and irrevocable enemies. The marriage, accomplished in spite of his mother’s vehement objections, was a weekend justice-of-the-peace affair.

The sexual experience of the courtship had been overwhelming to the physiologically and psychologically virginal male. The uninitiated man literally was seduced by the experienced woman, who manipulated, fellated, and coitally ejaculated him within three weeks of their initial meeting.

The hectic pace of the premarital sexual experience continued for the first 18 months of the marriage, with Mr. B awed by and made increasingly anxious by his wife’s sexual demands.

Intercourse occurred at least once a day. Following the pattern established during the courtship, opportunities, techniques, positions, procedures, durations, and recurrences, in fact, all sexual expression in the marriage, was at his wife’s able direction.

For the first year of the marriage the wife thoroughly enjoyed overwhelming her fully cooperative but naive and insecure husband with the force and frequency of her sexual demands. As the marriage continued unwavering in the intensity of her insistence upon sexual and social dominance, his confidence in his facility for sexual functioning began to wane.

He sought excuses to avoid coital connection, yet when cornered tried valiantly to respond to her demands. Finally, there were three occasions when sudden demand for coital connection forced failure of erection for the satiated male. Her comments were harsh and destructive, and the sarcasm struck a familiar chord.

The fourth time he failed to satisfy her immediate sexual needs, his wife’s denunciations reminded him specifically of his mother and of her verbal attacks on his father. For the first time in his life he identified with the man sitting in a corner of the living room reading the newspaper, and within a month’s time he had withdrawn to a similarly recessive behavioral patterning within his own home.

Successful erection
There is only one subsequent recorded episode of erection sufficiently successful for intromission with his wife. Aside from this, the man was totally impotent and had been so for three years when seen in therapy.

On an occasion when his wife was out of town, he followed the time-honored response pattern of the secondarily impotent man. There was attempted sex with a prostitute to see whether he could function effectively with any other woman.

For the first time in several months there was a full erection, but when he attempted to mount, the concept of his mother’s disapproval of his behavior disturbed his fantasy of female conquest. He immediately lost and could not recover the erection. This was his only attempt at extramarital sexual functioning.

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Secondary Impotence

Impotence Influence

An illustration of the repressive influence of religious orthodoxy upon the potential effectiveness of sexual functioning can be provided by relating the history of one of the five couples with both husband and wife products of different religious orthodoxies.

Impotence and Religion

Mr. and Mrs. D were married in their early twenties. He was the product of a fundamentalist Protestant background, she of equally strict Roman Catholic orientation. The man had the additional disadvantage of being an only child, while the wife was one of three siblings. The marriage was established over the firm and often expressed objections of both families.

Impotence and Sex Information

Prior to marriage the wife had no previous heterosexual, masturbatory, or homosexual history, and knew nothing of male or female sexual expression. She had been taught that the only reason for sexual functioning was for conceptive purposes.

Similarly, the husband had no exposure to sex information other than the vague directions of the peer group.

He had never seen a woman undressed either in fact or in pictures.

Dressing and toilet privacy had been the ironclad rule of the home. He also had been taught that sexual functioning could be condoned only if conception was desired.

His sexual history consisted of masturbation during his teenage years with only occasional frequency, and two prostitute exposures. He was totally unsuccessful in each exposure because he was presumed a sexually experienced man by both women.

Sex with Prostitute

During the first episode the prostitute took the unsuspecting virginal male to a vacant field and suggested they have intercourse while she leaned against a stone fence. Since he had no concept of female anatomy, of where to insert the penis, he failed miserably in this sexually demanding opportunity.

His graphic memory of the incident is of running away from a laughing woman.

Condom

The second prostitute provided a condom and demanded its use. He had no concept of how to use the condom. While the prostitute was demonstrating the technique, he ejaculated. He dressed and again fled the scene in confusion.

These two sexual episodes provided only anxiety-filled examples of sexual failure. Since he had no background from which to develop objectivity when considering his “sexual disasters,” inevitably the cultural misconception of lack of masculinity was the unfortunate residual of his experiences.

There was failure to consummate the marriage on the wedding night and for nine months thereafter. After consummation sexual function continued on a sporadic basis with no continuity. The wife refused contraception until after advent of the third child.

Sexual success was never of quality or quantity sufficient to relieve the husband of his fears of performance or to free the wife from the belief that either there was something wrong with her physically or that she was totally inadequate as a woman in attracting any man.

Sexual Difficulty

They rarely discussed their sexual difficulties, as both husband and wife were afraid of hurting one another, and each was certain that their unsatisfactory pattern of sexual dysfunction was all that could be expected from indulgence in sexual expression at times when conception was not the prime motivation.

With no appreciation of the naturalness of sexual functioning and with no concept of an honorable role for sexual response, the psychosocial pressures engendered from their negatively oriented sexual value systems left them with no positive means of mutual communication.

The failure of this marriage started with the wedding ceremony. There was no means of communication available for these two young people. Trained by theological demand to uninformed immaturity in matters of sexual connotation, both marital partners had no concept of how to cope when their sexual dysfunction was manifest. Their first approach to professional support was to agree to seek pastoral counseling.

Here their individual counselors were as handicapped by orthodoxy as were their supplicants. There were no suggestions made that possibly could have alleviated the sexual dysfunction. When sexual matters were raised, either no discussion was allowed, or every effort was made to belittle the importance of the sexual problem.

Without professional support, the marital partners were again released to their own devices. Each partner was intimidated, frustrated, and embarrassed for lack of sexual knowledge. The sexual dysfunction dominated the entire marriage.

The husband was never as effective professionally as he might have been otherwise. He withdrew from social functioning as much as possible. The wife was in a constant state of emotional turmoil, which had the usual rebound effect upon the children. By the time this husband and wife arrived at the Foundation, she was well on the way to earning the title of “shrew.”

Psycho-Sexual Performance

The couple was first seen after a decade of marriage. As expected from individuals so handicapped in communication, each partner had established an extramarital coital connection while individually searching for some security of personal identity and effectiveness of sexual performance.

The wife had been successful in establishing her own security of psycho-sexual performance; the husband, as would be anticipated in this instance, had not. After ten years of traumatic marriage, both individuals gravely questioned their religious beliefs. Although no longer channel visioned, the wife continued church attendance, the husband rejected all church affiliations.

There can be no feeling for naturalness of sexual expression when there is no background of sexual comprehension. There can be no appreciation that sexual functioning is indeed a natural physical phenomenon, when material of sexual content is considered overwhelmingly embarrassing, personally degrading, and often is theologically prohibited.

In essence, when an individual’s sexual value system has no positive connotation, how little the chance for truly effective sexual expression.

The fact that most men and woman survive the handicap of strict religious’ orthodoxy to function with some semblance of sexual effectiveness does not mean that these men and women are truly equipped to enjoy the uninhibited freedom of sexual exchange.

Their physical response patterns, developing in spite of their orthodox religious negation of an honorable role for sexual function, are immature, constrained, and, at times, even furtive.

Sexual function is stylized, unimaginative, depersonalized, and indeed productive only of biological reproduction. A derogatory affect upon the total personality is the tragic residual of conditioned inability to accept or handle objectively meaningful material of sexual content.

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Secondary Impotence

Impotence and Erection

It has such a varied etiology that a formalized frequency table for professional consideration is contraindicated at this time. Yet significant consideration must be devoted to dominant sources from which the fears of performance common to all forms of male sexual dysfunction can and do develop.

Every man is influenced to a major degree by his sexual value system, which reflects directly the input from his psychosocial background.

Over the centuries the single constant etiological source of all forms of male sexual dysfunction has been the level of cultural demand for effectiveness of male sexual performance. The cultural concept that the male partner must accept full responsibility for establishing successful coital connection has placed upon everyman the psychological burden for the coital process and has released every woman from any suggestion of similar responsibility for its success.

If anatomical anomalies such as vaginal agenesis or an imperforate hymen are exempted and the psychological dysfunction of vaginismus is discounted, it could be said provocatively that there has never been an impotent woman.

Woman need only make herself physically available to accomplish coital connection or even to propagate the race. Legions of women conceive and raise families without ever experiencing orgasm and carry coition to the point of male, ejaculation with little physical effort and no personal, reactive involvement.

During coition woman has only to lie still to be physically potent. While this role of total passivity is no longer an acceptable psychological approach to sexual encounter in view of current cultural demand for active female participation, it is still an irrevocable physiological fact that woman need only lie still to be potent.

Erection and masculinity

Any biophysical or psychosocial influence that can interfere with the male partner’s ability to achieve and to maintain an erection can cast a shadow of conscious doubt upon the effectiveness of his coital performance, and, in due course, upon his concept of the state of his masculinity.

Once a shadow of doubt has been cast, even though based only on a single unsatisfactory sexual performance after years of effective functioning, a man may become anxious about his theoretical potential for future coital connection. With the first doubt raised by any failed attempt at sexual connection in the past comes the first tinge of fear for the effectiveness of any sexual performance in the future.

There are a number of theoretical factors and a combination of psychological, circumstantial, environmental, physiological, or even iatrogenic factors that can raise the specter of the fear of performance in the always susceptible mind of the male in our culture, be he 14 or 84 years of age.

It should come as no surprise that in the referred population of sexually dysfunctional men, by far the most frequent potentiator of secondary impotence is the existence of a prior state of premature ejaculation, and that the second most frequent factor in onset of secondary impotence can be directly related to a specific incident of acute ingestion of alcohol or to a pattern of excessive alcohol intake per se.

Of course, both the factors of premature ejaculation and alcoholism accomplish their unfortunate purpose in the onset of impotence through engendering fears of performance.

In premature ejaculation
The fears of performance usually develop as the result of a slow but steady process of attrition spanning a period of years and are purely psychosocial in origin. In alcoholism the fears of performance usually develop rapidly, almost without warning, as the immediate result of untoward psychic trauma on circumstantial bases.

By reason of the diverse patterns of clinical onset as well as the marked variation in their rapidity of development, these two major etiological factors will be considered in some detail, with the discussion amplified by representative case histories.

Secondary Impotence With Premature Ejaculation

An established pattern of premature ejaculation prior to the onset of the symptoms of secondary impotence has been recorded in 63 of the total 213 men evaluated and treated for secondary impotence in the past 11 years. The premature ejaculation tendencies usually have been established for a significant period of time (generally a matter of years) before the symptoms of secondary impotence develop.

The fact that the prior existence of a pattern of premature ejaculation often leads to secondary impotence is yet another reason for clinical confusion in the textual listing of the premature ejaculator as an impotent male. There is no established percentage of premature ejaculators who progress to secondary impotence.

While the number is of considerable moment, this by no means suggests that a majority of premature ejaculators become secondarily impotent. A composite history typical of the sequential pattern of secondary impotence developing in a man distressed by prior symptoms of premature ejaculation is presented in detail.

Typically, the man is married, with some college education. Married in his mid-twenties, he usually is well into his thirties or even mid forties before onset of the symptoms of secondary impotence forces him to seek professional support.

Rapid ejaculations:

Sexual dysfunction (premature ejaculation) has existed throughout the marriage. This man has had a moderate degree of sexual experience before marriage with, perhaps, three to five other partners, and has the typical premature ejaculator’s history of having been conditioned in a rapid ejaculatory pattern during his first coital opportunities.

If authority has been approached in the interest of learning ejaculatory control, the results of such consultation have been essentially negligible in terms of improved sexual function. Professional relief of the psychosexual tensions created for the marital union by the continued existence of this form of sexual dysfunction rarely is sought until the youngest of any children of the marriage is at least of school age.

By this time the female partner has little tolerance for the situation. She no longer can contend with the frustrations inherent in a relatively constant state of sexual excitation, occasional, if ever, release of her sexual tensions, and rare, if ever, male consideration of her unresolved sexual demands.

Over the years of the marriage (ten to twenty), the issue of the husband’s rapid ejaculatory termination of their coital encounters has been raised repetitively.

The wife’s complaint was initially registered quietly, even questioningly; in time, complainingly or accusingly; and finally, demandingly, shrewishly, or contemptuously, as her personality and the immediate levels of her sexual frustration dictated.

The male partner, rarely made aware of the inadequacy of his sexual performances during premarital sexual experience, and frequently totally insensitive to his wife’s levels of sexual frustration during the early years of marriage, finally accepts the repetitively hammered concept that the dysfunctional state of their marital sexual status is “his fault” and, consequently, that he must “do something.”

And so he tries. As described in premature ejaculation, he bites his lips; thinks of work at the office; plans tomorrow’s activities; constricts the rectal sphincter; counts backwards from one hundred.

In short, does everything to distract himself from his partner’s obvious demands for sexual fulfillment during coital connection. Insofar as possible, he consciously turns off both the functional and the subjective projections of his wife’s sexual demands in order to reduce the input of his sexual stimuli.

Sexual Encounter

For instance whenever his wife reaches that level of sexual tension that finds her responding to sexually oriented stimuli almost involuntarily (a high-plateau tension level), the physically obvious state of her sexual demand drives her husband rapidly toward ejaculation. The beleaguered premature ejaculator, trying for control, employs any or all of the subjectively distracting tactics described above.

Thus, as much as possible, he not only denies the objective demand for his ejaculatory response inherent in his wife’s pelvic thrusting, but also attempts to deny generally the subjective feeling of vaginal containment and specifically the constrictive containment of the penis by her engorged orgasmic platform.

Insofar as possible, he compulsively negates the obvious commitment of her entire body to the elevated levels of her sexual demand. Whether or not this man ever acquires nominal physiological control of his premature ejaculatory tendencies by employing his diversionary tactics, one half of the mutually stimulative cycle that exists between sexually responsive men and women certainly has been dulled or even totally obviated.

This conscious dulling or even negating of input from his wife’s physical expressions of sexual demand is his first unintentional step toward secondary impotence.

There is marked individual variation in the particular moment at which the wife’s repetitively verbalized complaints of inadequacy of ejaculatory control were extrapolated by the husband into a conscious concern for “inadequacy of sexual performance.” Once the premature ejaculator develops any in depth concept that he is sexually inadequate, he is ripe for psychosocial distraction during any sexual encounter.

While his wife continues to berate his premature ejaculatory tendencies as “his sexual failure,” as “not getting the job done,” as “being totally uninterested in her sexual release,” or as “evidence of his purely selfish interests,” the reasonably intelligent male frequently develops a protean concern for the total of his sexual prowess.

Once a premature ejaculator questions the adequacy of his sexual performance, not only does he worry about ejaculatory control, but he also moves toward over concentration on the problem of satisfying his wife. While over concentrating in an attempt to force effective sexual control, he subjectively blocks full sensate input of the stimulative effect of his wife’s sexual demand.

Frequently, the pressured male resorts to a time honored female dodge: that of developing excuses for avoiding sexual activity. He claims he is tired not feeling well or has important work to do the next day.

He displays little interest in sexual encounter simply because he knows the result of any attempted sexual connection will probably be traumatic at best physical release for him but not satisfaction for his wife, and at worst a disaster of argument Or vituperation.

In brief:
There is further blocking of the inherent biophysical stimulation derived from the consistent level of mutual sexual awareness that prevails between sexually adjusted marital partners and a depreciation of the importance of mutual communication within the security of the marital bed.

Finally, the turning point. The wife pushes for sexual encounter on an occasion when the husband is emotionally distracted, physically tired, and certainly frustrated with his sexual failures. In a naturally self-protective sequence, he is totally uninterested in sexual encounter. When the husband is approached sexually by his demanding partner, there is little in the way of an erective response.

For the first time the man fears that he is dealing with a sexual dysfunction of infinitely more gravity than the performance inadequacy of his premature ejaculatory pattern. Once this man, previously sensitized to fears of sexual performance by his wife’s repetitively verbalized rejection of his rapid ejaculatory tendencies, fails at erection, fears of performance multiply almost geometrically, and his effectiveness as a sexually functional male diminishes with parallel rapidity.

Categories
Secondary Impotence

Impotence Cause by Surgery

In the second case, a supra-pubic prostatectomy, there was sufficient post surgical symptomatology to stimulate onset of symptoms of secondary impotence. In this situation the untoward surgical result was unfortunate. The distress in both instances was that the men had not been forewarned of the possible side effects of the surgery.

The case of secondary impotence developing after the supra-pubic prostatectomy was brought under control during therapy.

Prostatic cancer patients:
Those facing surgery, should be made aware by the operating surgeon that the loss of erective function can and does accompany such surgery. The psychosexual trauma forced upon the postoperative patient and his wife because they were not informed before surgery of the resultant sexual dysfunction is unforgivable.

The physiological influence of diabetes on secondary impotence is in a special category. In 6 of the total of 9 cases the onset of secondary impotence had been associated with the diabetes by consultative authority prior to referral for therapy, while in the remaining 3 instances no correlation between the established clinical condition of diabetes and the onset of impotence had been suggested by referring professionals.

Additionally, in 11cases of referral for secondary impotence without concept of etiological influence clinical diabetes (3 cases) and preclinical diabetes (8 cases) were diagnosed during metabolic work-ups that are part of the routine physical and laboratory evaluations of the secondarily impotent male referred for diagnosis and treatment.

As described in therapy treatments, a routine five-hour glucose-tolerance test is conducted for men referred for secondary impotence. This evaluation technique has been in effect for five years but has not reached the stage of statistical significance.

This work will be reported as a separate entity in monograph form at a later date. The statistical evaluation suggests that there is a 200-300 percent higher incidence of a diabetic or prediabetic curve reported for men with the clinical symptoms of secondary impotence, when returns are compared to the incidence of diabetic or prediabetic curves in similar glucose-tolerance testing of a representative cross-section of the population.

There is no supportable concept at this time that diabetes is an associate of equality with other etiological influences on secondary impotence. Nor does this work imply that the diabetic male has an established predisposition toward impotence. The amount of information available currently does not allow a firm clinical position.

Of course, there frequently are other etiological foci to combine with a diabetic or prediabetic state to influence the onset of secondary impotence. However, if a man is referred for secondary impotence, evaluation of his diabetic status should be a routine part of the total physical and laboratory work up.

It should be emphasized in context that even if symptoms of secondary impotence represent an end-point of etiological influence from a diabetic or prediabetic state, adequate institution and careful maintenance of medical control of the diabetes will not reverse the symptoms of impotence, once developed.

Impotence Diabetic

Difficulty lies, of course, in the fact that regardless of etiology, once lack of erective security has been established, fears of performance unalterably become an integral part of the psychosocial influences of the man’s daily life. Adequate medical control of the diabetes will provide no relief for his fears for sexual performance.

If diabetes or a prediabetic state can influence the onset of secondary impotence in other than advanced states of diabetic neuropathy, this fact is but another example of the multiple etiological aspects of secondary impotence.

Understandably, for many years the pattern of the human male has been to blame sexual dysfunction on specific physical distresses.

Every sexually inadequate male lunges toward any potential physical excuse for sexual malfunction. From point of ego support, would that it could be true.

A cast for a leg or a sling for an arm provides socially acceptable evidence of physical dysfunction of these extremities. Unfortunately the psychosocial causes of perpetual penile flaccidity cannot be explained or excused by devices for mechanical support.